Evidence mapPaperPMID 40465393Full record

ArticleCancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology2025

Breast Density Changes after Risk-Reducing Salpingo-oophorectomy in Women with a Pathogenic Germline Variant in BRCA1 or BRCA2.

Elizabeth A Loehrer, Frederieke H van der Baan, Thea M Mooij, Nadine Andrieu, Antonis C Antoniou, Monique D Dorrius, Douglas F Easton, Christoph Engel, Karin Kast, Ritse M Mann and 8 more

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In one paragraph

Article in Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

18 authors.

Elizabeth A Loehrer *Division of Molecular Pathology, Netherlands Cancer Institute, Amsterdam, the Netherlands.ORCID 0000-0003-1249-4442
Frederieke H van der Baan *Division of Psychological Research and Epidemiology, Netherlands Cancer Institute, Amsterdam, the Netherlands.ORCID 0000-0002-7421-6547
Thea M MooijDivision of Psychological Research and Epidemiology, Netherlands Cancer Institute, Amsterdam, the Netherlands.ORCID 0009-0009-3353-0693
Nadine AndrieuInserm U900, Paris, France.ORCID 0000-0001-8820-5550
Antonis C AntoniouCentre for Cancer Genetic Epidemiology, Department of Oncology, University of Cambridge, Cambridge, United Kingdom.ORCID 0000-0001-9223-3116
Monique D DorriusDepartment of Radiology, University Medical Center Groningen, Groningen, the Netherlands.ORCID 0000-0001-9825-6001
Douglas F EastonCentre for Cancer Genetic Epidemiology, Department of Oncology, University of Cambridge, Cambridge, United Kingdom.ORCID 0000-0003-2444-3247
Christoph EngelInstitute for Medical Informatics, Statistics and Epidemiology, Leipzig University, Leipzig, Germany.ORCID 0000-0002-7247-282X
Karin KastCenter for Hereditary Breast and Ovarian Cancer, Center for Integrated Oncology (CIO), Medical Faculty, University Hospital Cologne, Cologne, Germany.ORCID 0000-0001-6531-755X
Ritse M MannDepartment of Radiology, Netherlands Cancer Institute (Antoni van Leeuwenhoek), Amsterdam, the Netherlands.ORCID 0000-0001-8111-1930
Catherine NoguèsDepartment of Cancer Risk Management, Clinical Genetic Clinic, Institut Paoli-Calmettes, Marseille, France.ORCID 0009-0004-3940-9302
Rita K SchmutzlerCenter for Hereditary Breast and Ovarian Cancer, Center for Integrated Oncology (CIO), Medical Faculty, University Hospital Cologne, Cologne, Germany.ORCID 0000-0001-8160-4348
Yen Y TanDepartment of OB/GYN and Comprehensive Cancer Center, Medical University of Vienna, Vienna, Austria.ORCID 0000-0003-1063-5352
Mikael ErikssonDepartment of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.ORCID 0000-0001-8135-4270
Carla H van GilsDepartment of Epidemiology and Health Economics, University Medical Center Utrecht, Utrecht, the Netherlands.ORCID 0000-0003-0817-7567
Maartje J HooningDepartment of Medical Oncology, Erasmus MC Cancer Institute, Rotterdam, the Netherlands.ORCID 0000-0001-6763-0857
Matti A RookusDivision of Psychological Research and Epidemiology, Netherlands Cancer Institute, Amsterdam, the Netherlands.ORCID 0000-0001-9321-7919
Marjanka K SchmidtDivision of Molecular Pathology, Netherlands Cancer Institute, Amsterdam, the Netherlands.ORCID 0000-0002-2228-429X

Funding

Horizon 2020 Framework Programme (H2020) 964264KWF Kankerbestrijding (DCS) 12535
6 · The paper itself

Abstract

backgroundWe studied changes in mammographic density (MD) among premenopausal women with a pathogenic germline variant (PGV) in the BRCA1 or BRCA2 gene, comparing those who did and did not undergo risk-reducing salpingo-oophorectomy (RRSO) in the interval between mammograms, accounting for changes in exogenous oral contraceptive or hormone replacement therapy (HRT) use.

methodsFrom five studies of the International BRCA1/2 Carrier Cohort Study consortium, we included 691 participants who had two or more screening mammograms available, were less than 47 years at the time of RRSO (N = 208), or premenopausal at all mammograms without RRSO (N = 483). MD metrics [percent density (PD), dense area (DA), and non-DA] were quantified using STRATUS. Multivariable linear mixed models assessed changes in MD metrics between groups, adjusting for confounders.

resultsThe mean PD at first mammogram was 26.8% ± 15.3 (RRSO) and 31.3% ± 18.1 (no RRSO). In a median 1.1 years between mammograms, PD decreased on average by 0.9% [95% confidence interval (CI), -1.6 to -0.2] among women who did not undergo RRSO in the interval between mammograms compared with 5.9% (95% CI, -7.4 to -4.5) among women who underwent RRSO in the interval (adjusted difference, -5.9%; 95% CI, -9.5 to -2.2; P = 0.002). Results were driven primarily by MD changes among BRCA2 PGV carriers. The use of HRT after RRSO attenuated the decline in PD.

conclusionsOn average, PD and DA decrease following RRSO in premenopausal carriers, particularly among BRCA2 PGV carriers. HRT formulation affects MD changes. IMPACT: A decrease in MD may inform the potential protective effect of RRSO against breast cancer.

Indexed as

BRCA1 ProteinBRCA2 ProteinBreast DensityBreast NeoplasmsSalpingo-oophorectomyAdultFemaleGerm-Line MutationHumansMammographyMiddle AgedPremenopauseBRCA1 ProteinBRCA1 protein, humanBRCA2 ProteinBRCA2 protein, human

Identifiers

PMID40465393
PMCPMC12314511

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.