Evidence map›Paper›PMID 40481696›Full record

ArticleEuropean journal of clinical investigation2025

Gastric microbiota-specific signatures in adults with obesity and Helicobacter pylori-negative gastritis.

José Ignacio Martínez-Montoro, Raquel Sancho-Marín, Luis Ocaña-Wilhelmi, Isabel Arranz-Salas, Nerea Ruiz-Campos, María José García-López, Francisco J Tinahones, Carolina Gutiérrez-Repiso

Abstract read
In one paragraph

Article in European journal of clinical investigation, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

José Ignacio Martínez-MontoroInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.ORCID https://orcid.org/0000-0001-9761-6888
Raquel Sancho-MarínInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
Luis Ocaña-WilhelmiInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
Isabel Arranz-SalasInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
Nerea Ruiz-CamposInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
María José García-LópezInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
Francisco J TinahonesInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.
Carolina Gutiérrez-RepisoInstituto de Investigación Biomédica de Málaga-Plataforma en Nanomedicina (IBIMA-Plataforma Bionand), Málaga, Spain.

Funding

Centro de Investigación Biomédica en Red-Fisiopatología de la Obesidad y NutriciónEuropean Regional Development FundFunding for open access charge: Universidad de Málaga/CBUAInstituto de Salud Carlos III CM22/00217Instituto de Salud Carlos III CP20/00066Instituto de Salud Carlos III JR24/00006Ministerio de Ciencia e Innovación PTA2022-021854-I
6 · The paper itself

Abstract

backgroundThe role of the gastric microbiome in the pathophysiology of gastritis beyond Helicobacter pylori (HP) infection is poorly understood and has remained unexplored in patients with obesity. The aim of this study was to analyse gastric mucosa-associated microbiota in patients with obesity and nonatrophic chronic gastritis in the absence of HP infection or history of HP eradication.

methodsThis was a case-control study conducted at Virgen de la Victoria University Hospital in Malaga, performed in patients with severe obesity (body mass index ≥40 kg/m

resultsSixty-seven participants (38 in the gastritis and 29 in the nongastritis group) were included. A lower alpha diversity (evenness and Shannon diversity indexes) and beta diversity (weighted Unifrac distance) were shown in the gastritis group. Higher relative abundances in the families Micrococcaceae, Streptococcaceae and Leuconostocaceae and the genera Streptococcus, Weissella and Cryptobacterium were observed in the gastritis group, compared with the nongastritis group. An enrichment in pathways involved in toluene degradation, heterolactic fermentation and secondary metabolites biosynthesis, such as ergothioneine and terpenoids, was found in the gastritis group. Also, higher total cholesterol levels 1 year after the surgery were observed in the gastritis group compared with the nongastritis group, although no within-group differences from baseline to 1 year were detected in this parameter.

conclusionOur results suggest a relationship between the gastric microbiome and nonatrophic chronic gastritis in obesity, beyond HP infection.

Indexed as

Gastric MucosaGastritisGastrointestinal MicrobiomeObesityObesity, MorbidAdultCase-Control StudiesChronic DiseaseFemaleGastrectomyHelicobacter InfectionsHelicobacter pyloriHumansMaleMiddle AgedProspective StudiesRNA, Ribosomal, 16S16S rRNAgastric microbiotanonatrophic chronic gastritisobesity

Identifiers

PMID40481696
PMCPMC12517247

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.