Evidence map›Paper›PMID 40524287›Full record

ArticleJournal of traditional Chinese medicine = Chung i tsa chih ying wen pan2025

Matrine alleviates coronary microvascular dysfunction in ischemia with non-obstructive coronary artery disease mice induced by advanced glycation end products inhibition of the reactive oxygen species-mediated endoplasmic reticulum stress in cardiac microvascular endothelial cells.

D U Haixia, Qiu Chuan, M A Yanpeng, Pan Shuo, Wang Xiqiang, Wang Junkui, Liu Zhongwei

Abstract read
In one paragraph

Article in Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
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1 · What the graph read from it

What it found

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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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0 citing papers in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

7 authors.

D U HaixiaDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.
Qiu ChuanDivision of Biomedical Informatics and Genomics, Deming Department of Medicine, Tulane Center of Biomedical Informatics and Genomics, Tulane University, New Orleans 70112, USA.
M A YanpengDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.
Pan ShuoDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.
Wang XiqiangDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.
Wang JunkuiDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.
Liu ZhongweiDepartment of Cardiology, Shaanxi Provincial People's Hospital, Xi'an 710068, China.

Funding

National Natural Scientific Foundation of China: Mechanisms of Macrophage-Mediated Vascular Smooth Muscle Cells Phenotypic Conversion in Advanced Glycation End Products-induced Atherosclerosis and Therapeutic Effects of Targeted Gene Silencing 82070858Youth Scientific Research and Innovation Team Program of Shaanxi Province: Diabetes-Related Atherosclerosis Basic Research and Application Research Team 2022-SLRH-LJ-014
6 · The paper itself

Abstract

objectiveTo investigate the protective effect of matrine on coronary microvascular dysfunction (CMD) induced by advanced glycation end products (AGEs) in a mouse model of ischemia with non-obstructive coronary artery disease (INOCA), with a focus on the underlying mechanisms, particularly the endoplasmic reticulum (ER) stress protein kinase R-like ER kinase (PERK)/ nuclear factor of activated T-cells (NFAT) signaling pathway.

methodsAn INOCA model was established in mice, and CMD was induced by peritoneal injections of AGEs. Matrine was administered daily

resultsMatrine treatment significantly improved CFVR and reduced CMD in AGEs-exposed INOCA mice. In CMECs, matrine attenuated AGEs-induced apoptosis, inflammation, and microthrombosis. It also suppressed intracellular reactive oxygen species (ROS) generation, ER stress markers, and PERK/NFAT signaling. Matrine's effects were concentration-dependent and partially reversed by the PERK agonist, confirming its action through the ER stress pathway. No significant toxicities were observed with matrine administration.

conclusionMatrine attenuates AGEs-induced CMD in INOCA by suppressing the ROS-mediated ER stress PERK/NFAT signaling pathway in CMECs. This study highlights matrine's potential as a therapeutic agent for CMD in diabetic cardiovascular complications.

Indexed as

AlkaloidsCoronary Artery DiseaseEndoplasmic Reticulum StressEndothelial CellsGlycation End Products, AdvancedQuinolizinesReactive Oxygen SpeciesAnimalsApoptosisCoronary VesselsHumansMaleMatrinesMiceMice, Inbred C57BLMicrocirculationAlkaloidsGlycation End Products, AdvancedMatrinesQuinolizinesReactive Oxygen Speciescardiac microvascular endothelial cellscoronary microvascular dysfunctionendoplasmic reticulum stressglycation end products, advancedmatrine

Identifiers

PMID40524287
PMCPMC12134324

What Socratic holds

Textmetadata
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.