Evidence map›Paper›PMID 40534259›Full record

ArticleAlzheimer's & dementia : the journal of the Alzheimer's Association2025

Increased activity of Piezo1 channel in red blood cells is associated with Alzheimer's disease-related dementia.

Valeriia Sitnikova, Dilyara Nurkhametova, Nicoletta Braidotti, Catalin D Ciubotaru, Luca Giudice, Ulla Impola, Sari Kärkkäinen, Juho Kalapudas, Elina Penttilä, Heikki Löppönen and 9 more

Abstract read
In one paragraph

Article in Alzheimer's & dementia : the journal of the Alzheimer's Association, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Article
  2. Review
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  4. Review
  5. Review
  6. Increased activity of Piezo1 channel in red blood cells is associated with Alzheimer's disease-related dementia.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2025
    Article
  7. Review
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

19 authors.

Valeriia SitnikovaA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.ORCID 0000-0002-8192-1236
Dilyara NurkhametovaA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.
Nicoletta BraidottiMaterials Foundry, National Research Council of Italy (CNR-IOM), Area Science Park Basovizza, Trieste, Italy.
Catalin D CiubotaruMaterials Foundry, National Research Council of Italy (CNR-IOM), Area Science Park Basovizza, Trieste, Italy.
Luca GiudiceA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.
Ulla ImpolaResearch and Development, Finnish Red Cross Blood Service, Helsinki, Finland.
Sari KärkkäinenBiomarker Laboratory, Institute of Clinical Medicine/Neurology, University of Eastern Finland, Kuopio, Finland.
Juho KalapudasNeuro Centre, Kuopio University Hospital, Kuopio, Finland.
Elina PenttiläDepartment of Otorhinolaryngology, School of Medicine, University of Eastern Finland, and Kuopio University Hospital, Kuopio, Finland.
Heikki LöppönenDepartment of Otorhinolaryngology, School of Medicine, University of Eastern Finland, and Kuopio University Hospital, Kuopio, Finland.
Ilkka FagerlundA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.
Katja M KanninenA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.
Henrik ZetterbergClinical Neurochemistry Laboratory, Sahlgrenska University Hospital, Göteborg, Sweden.
Tarja KokkolaDepartment of Geriatrics and Neurological Memory polyclinic, Helsinki University Hospital and Neurosciences, Faculty of Medicine, University of Helsinki, Helsinki, Finland.
Saara LaitinenResearch and Development, Finnish Red Cross Blood Service, Helsinki, Finland.
Anne KoivistoNeuro Centre, Kuopio University Hospital, Kuopio, Finland.
Dan CojocMaterials Foundry, National Research Council of Italy (CNR-IOM), Area Science Park Basovizza, Trieste, Italy.
Rashid GiniatullinA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.
Tarja M MalmA.I.  Virtanen Institute for Molecular Sciences, University of Eastern Finland, Kuopio, Finland.

Funding

Academy of FinlandBusiness FinlandHORIZON EUROPE European Research Council 101053962Swedish Research Council 2019-02397Swedish Research Council 2022-01018Swedish Research Council 2023-00356Swedish State Support for Clinical Research ALFGBG-71320
6 · The paper itself

Abstract

introductionRed blood cells (RBCs) are crucial for oxygen delivery to active tissues and endure significant mechanical forces in the microcirculatory bed. The enrichment of mechanosensitive Piezo1 channels, linked to the cytoskeleton, aids RBCs in navigating the narrow capillaries. In Alzheimer's disease (AD), impaired brain microcirculation may necessitate enhanced Piezo1 function in RBCs.

methodsWith micropipette aspiration and flow cytometry technics, we evaluated, using the specific Piezo1 agonist Yoda1, AD-related alterations in the biomechanical properties of RBCs from cognitively healthy patients (HC) and individuals with mild cognitive impairment (MCI) and AD.

resultsWe show that beta-amyloid (Aβ) peptides alter the biomechanical properties of RBCs. We observed significantly higher Yoda1-induced calcium responses in RBCs in individuals with MCI and AD compared to RBCs from age-matched HC.

conclusionOur data suggest that Yoda1-induced Ca HIGHLIGHTS: Piezo1 channels aid the navigation of red blood cells (RBCs) through narrow capillaries. Alzheimer's disease (AD) patients show increased Yoda1-induced activation of Piezo1 in RBCs. Incorporation of Yoda1-induced Piezo1 readouts improved the detection of AD-related dementia. Investigating Yoda1-induced Piezo1 activity associated with early AD.

Indexed as

Alzheimer DiseaseCognitive DysfunctionErythrocytesIon ChannelsAgedAged, 80 and overAmyloid beta-PeptidesCalciumFemaleHumansMalePyrazinesThiadiazolesAmyloid beta-PeptidesCalciumIon ChannelsPIEZO1 protein, humanPyrazinesThiadiazolesyoda-1Alzheimer's diseaseCa‐imagingPiezo1RBC

Identifiers

PMID40534259
PMCPMC12177196

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.