Evidence mapPaperPMID 40535646Full record

ReviewAmerican journal of translational research2025

Role of gut-brain axis dysregulation in the pathogenesis of non-alcoholic fatty liver disease: mechanisms and therapeutic implications.

Mei Wan, Weimin Zhao, Yifan Cai, Qian He, Jun Zeng

Abstract readReview
In one paragraph

Review in American journal of translational research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

Mei WanThe First Clinical Medical College of China Three Gorges University, Department of Endocrinology, Institute of Endocrine and Metabolic Diseases, Yichang Central People's Hospital Yichang 443000, Hubei, China.
Weimin ZhaoThe First Clinical Medical College of China Three Gorges University, Department of Endocrinology, Institute of Endocrine and Metabolic Diseases, Yichang Central People's Hospital Yichang 443000, Hubei, China.
Yifan CaiLife Science, Queen's University Kingston K7L 3N6, Ontario, Canada.
Qian HeLaboratory of Metabolic Bone Diseases, and Department of Metabolism and Endocrinology, The Second Xiangya Hospital of Central South University Changsha 410011, Hunan, China.
Jun ZengThe First Clinical Medical College of China Three Gorges University, Department of Endocrinology, Institute of Endocrine and Metabolic Diseases, Yichang Central People's Hospital Yichang 443000, Hubei, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Non-alcoholic fatty liver disease (NAFLD) has emerged as a global health challenge due to its rising prevalence and strong association with metabolic syndrome. Recent studies highlight the critical role of the gut-brain axis (GBA)-a bidirectional communication system between the gut, brain, and liver-in NAFLD pathogenesis. Dysregulation of this axis can worsen metabolic dysfunction, inflammation, and liver injury. This review discusses the mechanisms driving GBA dysregulation in NAFLD, including alterations in gut microbiota, increased intestinal permeability, neuroinflammation, and imbalances in the autonomic nervous system (ANS). We also explore therapeutic strategies, such as microbiota modulation, vagus nerve stimulation, and neuroprotective interventions, that may help mitigate the effects of GBA dysfunction on NAFLD progression.

Indexed as

Gut-brain axis dysregulationmechanismsnon-alcoholic fatty liver diseasetherapeutic implications

Identifiers

PMID40535646
PMCPMC12170361

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.