ArticleAdvanced science (Weinheim, Baden-Wurttemberg, Germany)2025
Intestinal Clock Promotes Cognitive Memory Through Adenosine Signaling.
Article in Advanced science (Weinheim, Baden-Wurttemberg, Germany), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
4 citing papers in PubMed.
- Disease-stage-specific immunometabolic remodeling in pediatric obstructive sleep apnea: a single-cell transcriptomic atlas of adenoid tissue.EMBO molecular medicine · 2026Article
- Stachyose supplementation attenuates ischemic stroke injury via gut-brain axis mediated adenine metabolism and neuroactive ligand-receptor signaling.Current research in food science · 2026Article
- Artificial intelligence empowers gut microbiota research in neurodegenerative diseases molecular mechanisms and precision therapy.iScience · 2025Review
- Intestinal Clock Promotes Cognitive Memory Through Adenosine Signaling.Advanced science (Weinheim, Baden-Wurttemberg, Germany) · 2025Article
Corrections and comments
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Authors and funding
14 authors.
Funding
Abstract
Although the intestinal clock (the circadian timing system in the gastrointestinal tract) is known to direct a wide variety of diurnal nutrients and metabolites, its role in the functioning of extra-intestinal tissues such as the brain remains elusive. Here the role of the intestinal clock in shaping cognitive function is investigated. It is found that Bmal1-iKO mice (mice with Bmal1 [Brain and muscle Arnt-like protein 1] specifically knocked out in the intestine, a mouse line deficient in intestinal clock function) show a defect in cognitive memory irrespective of the time-of-day. Bmal1-iKO-associated cognitive decline is attributed to impaired adenosine signaling and compromised long-term potentiation (LTP) in the hippocampus. Adenosine signaling promotes LTP via enhancing BDNF expression and inhibiting synapse loss. Furthermore, the impairment in adenosine signaling is accounted for by the reductions in intestinal absorption of and hippocampal level of adenosine but not by a change in adenosine receptors. Consistently, adenosine supplementation rescues cognitive deficits associated with the malfunction of the intestinal clock. Moreover, BMAL1 regulates the expression of ADK (adenosine kinase, a primary enzyme for adenosine clearance) in the small intestine and thus promotes intestinal adenosine absorption through REV-ERBα which binds directly to Adk P2 promoter to inhibit its transcription. Together, an unsuspected role of the intestinal clock in controlling cognitive memory is identified, highlighting the intestinal clock as a promising target for the management of cognitive disorders.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.