Evidence mapPaperPMID 40559667Full record

ReviewMarine drugs2025

Targeting Ferroptosis in Tumors: Novel Marine-Derived Compounds as Regulators of Lipid Peroxidation and GPX4 Signaling.

Yimao Wu, Xiaoyan Chen, Zichang Chen, Yunqi Ma

Abstract readReview
In one paragraph

Review in Marine drugs, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
18citing papers in PubMed, 2 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

18 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Interaction of HIF-1a with various cell death pathways in tumor immune microenvironment (TIME).Apoptosis : an international journal on programmed cell death · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Yimao WuSchool of Pharmacy, Binzhou Medical University, Yantai 264003, China.ORCID 0009-0004-4116-2687
Xiaoyan ChenSecond Clinical Medical College, Guangdong Medical University, Dongguan 523808, China.
Zichang ChenSecond Clinical Medical College, Guangdong Medical University, Dongguan 523808, China.
Yunqi MaSchool of Pharmacy, Binzhou Medical University, Yantai 264003, China.ORCID 0009-0006-9798-8060

Funding

Binzhou Medical University Research Fund Project BY2021KYQD02
6 · The paper itself

Abstract

This article reviews the mechanisms by which marine natural products regulate ferroptosis and their potential applications in tumor therapy. Ferroptosis is a form of programmed cell death driven by iron-dependent lipid peroxidation, characterized primarily by the accumulation of lipid peroxides and the failure of antioxidant defense systems. Due to their unique chemical structural diversity, marine natural products demonstrate significant advantages in regulating the ferroptosis pathway. Studies showed that marine compounds target key molecules such as glutathione peroxidase 4 (GPX4) and long-chain acyl-CoA synthetase 4 (ACSL4(a)) ACSL4(1) to modulate lipid peroxidation and iron metabolism, inducing ferroptosis in tumor cells and reshaping the tumor microenvironment (TME). In addition, marine compounds can enhance anti-tumor effects by activating immune responses. Although marine compounds hold great potential in regulating ferroptosis, their clinical translation faces challenges such as low bioavailability and tumor type dependency. Future research needs to integrate multi-omics techniques to further analyze the mechanisms of marine compounds and develop precision therapeutic strategies based on marine compounds to overcome the bottlenecks in ferroptosis therapy.

Indexed as

Antineoplastic AgentsAquatic OrganismsBiological ProductsFerroptosisLipid PeroxidationNeoplasmsPhospholipid Hydroperoxide Glutathione PeroxidaseAnimalsHumansSignal TransductionTumor MicroenvironmentAntineoplastic AgentsBiological ProductsPhospholipid Hydroperoxide Glutathione PeroxidaseACSL4(1)ferroptosisGPX4lipid metabolismlipid peroxidationmarine-derived compoundstargeted therapy

Identifiers

PMID40559667
PMCPMC12194754

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.