Evidence mapPaperPMID 40563367Full record

ReviewAntioxidants (Basel, Switzerland)2025

Oxidative Stress and Inflammation: Drivers of Tumorigenesis and Therapeutic Opportunities.

Meimei Wang, Yaping Xiao, Jie Miao, Xin Zhang, Meng Liu, Longchao Zhu, Hongxin Liu, Xiaoyan Shen, Jihui Wang, Biao Xie and 1 more

Abstract readReview
In one paragraph

Review in Antioxidants (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 52 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
52citing papers in PubMed, 2 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

52 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. Pooled it
  3. Article
  4. Article
  5. Metabolomic, in vitro and preclinical evaluation of Auricularia polytricha mushroom.International microbiology : the official journal of the Spanish Society for Microbiology · 2026
    Article
  6. Review
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  11. Article
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  15. Article
  16. Mature-StageInternational journal of molecular sciences · 2026
    Article
  17. Article
  18. Article
  19. Article
  20. The Oxidative Stress: Origin and Role in Aging and Diseases.Antioxidants (Basel, Switzerland) · 2026
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Meimei WangSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Yaping XiaoSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Jie MiaoSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Xin ZhangSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Meng LiuSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Longchao ZhuSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Hongxin LiuSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Xiaoyan ShenSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Jihui WangSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.
Biao XieDepartment of Gastroenterology, Guangzhou Eighth People's Hospital, Guangzhou Medical University, Guangzhou 510440, China.ORCID 0000-0002-2572-6882
Di WangSchool of Life and Health Technology, Dongguan University of Technology, Dongguan 523808, China.

Funding

Guangdong basic and applied basic research foundation 2023A1515140175National Natural Science Foundation of China 32301201National Natural Science Foundation of China 82100586Shenzhen Science and Technology Program JCYJ20220530165205012
6 · The paper itself

Abstract

As two pivotal regulatory factors in cancer biology, oxidative stress and inflammation interact dynamically through complex network mechanisms to influence tumor initiation, progression, and treatment resistance. Oxidative stress induces genomic instability, oncogenic signaling activation, and tumor microenvironment (TME) remodeling via the abnormal accumulation of reactive oxygen species (ROS) or reactive nitrogen species (RNS). Conversely, inflammation sustains malignant phenotypes by releasing pro-inflammatory cytokines and chemokines and promoting immune cell infiltration. These processes create a vicious cycle via positive feedback loops whereby oxidative stress initiates inflammatory signaling, while the inflammatory milieu further amplifies ROS/RNS production, collectively promoting proliferation, migration, angiogenesis, drug resistance, and immune evasion in tumor cells. Moreover, their crosstalk modulates DNA damage repair, metabolic reprogramming, and drug efflux pump activity, significantly impacting the sensitivity of cancer cells to chemotherapy, radiotherapy, and targeted therapies. This review systematically discusses these advances and the molecular mechanisms underlying the interplay between oxidative stress and inflammation in cancer biology. It also explores their potential as diagnostic biomarkers and prognostic indicators and highlights novel therapeutic strategies targeting the oxidative stress-inflammation axis. The goal is to provide a theoretical framework and translational roadmap for developing synergistic anti-tumor therapies.

Indexed as

cancer biologyinflammationoxidative stresstumor therapies

Identifiers

PMID40563367
PMCPMC12189506

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.