Evidence map›Paper›PMID 40565262›Full record

ArticleInternational journal of molecular sciences2025

Integrated Multi-Omics Analysis Reveals the Mechanisms of Intestinal Cell Injury Under Different Levels of Heat Stress.

Yuchao Feng, Decheng Suo, Ping Gong, Peiling Wei, Lu Zhang, Shu Zhang, Xiaonan Li, Changyuan Wang, Xia Fan

Abstract read
In one paragraph

Article in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Yuchao FengState Key Laboratory for Quality and Safety of Agro-Products, Institute of Quality Standards and Testing Technology for Agro-Products, Chinese Academy of Agricultural Sciences, Beijing 100081, China.
Decheng SuoState Key Laboratory for Quality and Safety of Agro-Products, Institute of Quality Standards and Testing Technology for Agro-Products, Chinese Academy of Agricultural Sciences, Beijing 100081, China.
Ping GongInstitute of Animal Husbandry Quality Standards, Xinjiang Academy of Animal Science, Urumqi 830057, China.
Peiling WeiInstitute of Animal Husbandry Quality Standards, Xinjiang Academy of Animal Science, Urumqi 830057, China.
Lu ZhangCenter of Agro-Product Safety and Quality of Xinjiang, Urumqi 830006, China.
Shu ZhangCollege of Food, Heilongjiang Bayi Agricultural University, Daqing 163319, China.
Xiaonan LiState Key Laboratory for Quality and Safety of Agro-Products, Institute of Quality Standards and Testing Technology for Agro-Products, Chinese Academy of Agricultural Sciences, Beijing 100081, China.ORCID 0009-0007-4913-2572
Changyuan WangCollege of Food, Heilongjiang Bayi Agricultural University, Daqing 163319, China.
Xia FanState Key Laboratory for Quality and Safety of Agro-Products, Institute of Quality Standards and Testing Technology for Agro-Products, Chinese Academy of Agricultural Sciences, Beijing 100081, China.ORCID 0009-0006-4421-1530

Funding

Ministry of Science and Technology of the People's Republic of China [DQKLLYD001]Science and Technology Department of Heilongjiang [2022LQ01002-02]Science and Technology Department of Heilongjiang [2022ZX02B18]
6 · The paper itself

Abstract

Given the escalating global temperatures and the consequent exacerbation of heat stress, dietary interventions have emerged as a promising therapeutic strategy. The gastrointestinal tract, being exquisitely sensitive to thermal challenges, revealing the underlying mechanisms of intestinal cell injury under high temperature, is essential for developing strategies to prevent heat stress. Here, we integrated metabolomic and transcriptomic analyses to investigate the metabolic and genetic changes in murine intestinal cells in response to different levels of heat stress. The results identified the PI3k-Akt-FoxO pathway as the major heat stress regulatory pathway Kin MODE-K cells. The possible regulatory mechanism is to reduce the expression of the

Indexed as

Heat-Shock ResponseIntestinal MucosaIntestinesMetabolomicsAnimalsApoptosisForkhead Transcription FactorsGene Expression ProfilingGene Expression RegulationMiceMultiomicsPhosphatidylinositol 3-KinasesProto-Oncogene Proteins c-aktSignal TransductionTranscriptomeForkhead Transcription FactorsPhosphatidylinositol 3-KinasesProto-Oncogene Proteins c-aktheat stressmechanismsmetabolomicspathwaytranscriptomics

Identifiers

PMID40565262
PMCPMC12192742

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.