Evidence map›Paper›PMID 40580368›Full record

ReviewJournal of molecular histology2025

The role of the CXC chemokine family in endometriosis research and its therapeutic potential.

Yan Deng, Qin Liu

Abstract readReview
PubMed Publisher
In one paragraph

Review in Journal of molecular histology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Yan DengSchool of Medicine, Jiangsu University, Zhenjiang, Jiangsu, China.
Qin LiuDepartment of Gynecology, Affiliated Kunshan Hospital of Jiangsu University, Jiangsu University, No.566 Qianjin East Rd., Kunshan, Suzhou, 215300, Jiangsu, China. tongzhanzhui4243@163.com.

Funding

Obstetrics and Gynecology Clinical and Basic Research Team- The First People's Hospital Of Kunshan Institutional Medical Education Advancement CXTD21-C01
6 · The paper itself

Abstract

Endometriosis (EM) is a chronic estrogen-dependent gynecological disease that affects approximately 10% of women of reproductive age and is a leading cause of infertility worldwide. In addition to reproductive dysfunction, EM is associated with chronic pain, psychological distress, and significant healthcare costs, yet its etiopathogenesis remains incompletely understood. Chemokines, especially members of the CXC family, have attracted increasing attention for their roles in EM-related inflammation, immune dysregulation, cell migration, angiogenesis, and pain modulation. These small signaling proteins, typically 8-10 kDa in size, are aberrantly expressed in ectopic lesions and participate in both disease initiation and progression. This review aims to systematically summarize current research on the expression patterns and biological functions of CXC chemokines in EM, with a particular focus on their contributions to diagnostic strategies, pathological mechanisms, and therapeutic development. While emerging evidence highlights their importance, further studies are needed to clarify subtype-specific mechanisms and clinical applicability. Understanding the roles of CXC chemokines may help uncover novel molecular targets for diagnosis and intervention, thereby advancing more effective and personalized treatments for patients with endometriosis.

Indexed as

Chemokines, CXCEndometriosisAnimalsFemaleHumansChemokines, CXCChemokinesCXC familyEndometriosisGynecological

Identifiers

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.