Evidence mapPaperPMID 40585349Full record

ReviewInternational journal of hypertension2025

Mechanisms and Prevention Strategies of Macrophage Involvement in the Progression From Hypertension to Heart Failure.

Ningning Zhang, Pengyu Cao, Bojian Wang, Jinting Yang, Lijing Zhao, Wangshu Shao

Abstract readReview
In one paragraph

Review in International journal of hypertension, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Ningning ZhangThe Cardiovascular Center, Changzhou No. 2 People's Hospital, The Third Affiliated Hospital of Nanjing Medical University, Changzhou 213164, Jiangsu, China.
Pengyu CaoThe Cardiovascular Center, Changzhou No. 2 People's Hospital, The Third Affiliated Hospital of Nanjing Medical University, Changzhou 213164, Jiangsu, China.ORCID https://orcid.org/0000-0003-0560-5159
Bojian WangSchool of Nursing, Jilin University, Changchun 130021, Jilin, China.ORCID https://orcid.org/0009-0009-3935-3448
Jinting YangSchool of Nursing, Jilin University, Changchun 130021, Jilin, China.
Lijing ZhaoSchool of Nursing, Jilin University, Changchun 130021, Jilin, China.
Wangshu ShaoThe Cardiovascular Center, First Hospital of Jilin University, Changchun 130021, Jilin, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hypertensive heart disease, a condition affecting millions worldwide, encompasses a spectrum from uncontrolled hypertension to heart failure. Despite the intricacies of its pathogenic mechanisms, recent attention has been directed toward the role of macrophages in the progression from hypertension to heart failure. Under normal circumstances, macrophages contribute to tissue homeostasis by clearing deceased cells. However, dysregulation during hypertension triggers inflammatory pathways, leading to tissue damage. Oxidative stress and mitochondrial dysfunction are implicated in this process. Exercise training, gaining popularity for its potential in regulating macrophage function, emerges as a promising intervention to improve outcomes in hypertensive heart disease. This review provides a succinct overview of previous research elucidating the involvement of macrophages in the transition from hypertension to heart failure. It underscores the current active areas of investigation and emphasizes the potential of exercise training in mediating macrophage responses, offering a glimpse into a hopeful avenue for therapeutic intervention in this challenging medical condition.

Indexed as

exercise trainingheart failurehypertensionmacrophagesventricular remodeling

Identifiers

PMID40585349
PMCPMC12204743

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.