Evidence map›Paper›PMID 40616709›Full record

ArticleBiological trace element research2025

Linalool May Exert Neuroprotective Effects Against Cadmium-Induced Hippocampal Neurodegeneration by Regulating the 4-HNE/NF-κB Signaling Pathway.

Sercan Kaya, Tuba Yalçın, Devran Ayyıldız, Buşra Akyol

Abstract read
In one paragraph

Article in Biological trace element research, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Article
  2. Review
  3. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Sercan KayaVocational Higher School of Healthcare Studies, Health Services Vocational School, Batman University, Room 212, Kültür Neighborhood, Batman, Turkey. sercan.kaya@batman.edu.tr.ORCID http://orcid.org/0000-0001-9014-2448
Tuba YalçınVocational Higher School of Healthcare Studies, Health Services Vocational School, Batman University, Room 212, Kültür Neighborhood, Batman, Turkey.ORCID http://orcid.org/0000-0002-2359-9832
Devran AyyıldızVocational Higher School of Healthcare Studies, Health Services Vocational School, Batman University, Room 212, Kültür Neighborhood, Batman, Turkey.ORCID http://orcid.org/0000-0002-8244-7572
Buşra AkyolVocational Higher School of Healthcare Studies, Health Services Vocational School, Batman University, Room 212, Kültür Neighborhood, Batman, Turkey.ORCID http://orcid.org/0000-0001-5903-3905

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cadmium (Cd) is a prevalent industrial and environmental contaminant that accumulates in the tissues of animals and humans. In particular, one of the regions most affected by Cd-induced neurodegeneration is the hippocampus. Linalool (Lin) is a monoterpene capable of traversing the blood-brain barrier owing to its low molecular weight and high lipophilicity. This study sought to establish the possible neuroprotective effects of Lin against Cd-induced hippocampal neurodegeneration by focusing on the 4-hydroxynonenal (4-HNE)/Nuclear factor kappa B (NF-κB) signaling pathway. The 28 male rats used in the experiment were randomly (n = 7) distributed into four groups. The control group was not administered. The Cd group received CdCl

Indexed as

AldehydesCadmiumHippocampusMonoterpenesNeuroprotective AgentsNF-kappa BSignal TransductionAcyclic MonoterpenesAnimalsMaleOxidative StressRatsRats, Wistar4-hydroxy-2-nonenalAcyclic MonoterpenesAldehydesCadmiumlinaloolMonoterpenesNeuroprotective AgentsNF-kappa B4-hydroxynonenalCadmiumHippocampusLinaloolNuclear factor kappa B

Identifiers

PMID40616709
PMCPMC12602611

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.