Evidence mapPaperPMID 40630101Full record

ReviewFrontiers in endocrinology2025

Adipose tissue dysfunction disrupts metabolic homeostasis: mechanisms linking fat dysregulation to disease.

Dana Bou Matar, Mahmoud Zhra, Walid Khaled Nassar, Haifa Altemyatt, Asfiya Naureen, Nada Abotouk, Muhammad Affan Elahi, Ahmad Aljada

Abstract readReview
In one paragraph

Review in Frontiers in endocrinology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers, 3 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed, 3 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed, 3 syntheses or guidelines pooled it.

  1. Pooled it
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  6. The Sirtuin Network: Linking NADDiabetes/metabolism research and reviews · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Dana Bou MatarDepartment of Physiology, College of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Mahmoud ZhraDepartment of Biochemistry and Molecular Medicine, College of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Walid Khaled NassarCollege of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Haifa AltemyattCollege of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Asfiya NaureenCollege of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Nada AbotoukCollege of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Muhammad Affan ElahiCollege of Medicine, Alfaisal University, Riyadh, Saudi Arabia.
Ahmad AljadaDepartment of Biochemistry and Molecular Medicine, College of Medicine, Alfaisal University, Riyadh, Saudi Arabia.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Metabolic disease incidence continues rising globally. Adipose tissue dysfunction serves as a crucial pathophysiological mediator. We evaluate molecular mechanisms linking adipose dysfunction to metabolic dysregulation. Methods: We systematically reviewed literature on adipose biology, stress mechanisms, inflammation, and metabolic networks. Analysis prioritized methodologically robust studies from the past decade. Results: Adipose dysfunction disrupts metabolic homeostasis through complex molecular networks. Stressed adipocytes exhibit mitochondrial impairment and endoplasmic reticulum (ER) stress. These changes alter inflammatory mediators and adipokine secretion. Brown and beige adipose regulate energy balance via uncoupling protein 1 (UCP1)-mediated thermogenesis. Key transcriptional regulators, PGC-1α and PR domain containing 16 (PRDM16), control thermogenic adipocyte development. Cellular senescence contributes significantly to age-related adipose dysfunction through inflammatory secretory phenotypes. Brown fat also secretes specialized factors influencing whole-body metabolism, emphasizing adipose tissue's endocrine function. Conclusion: Adipose dysfunction represents a critical nexus in metabolic disease pathogenesis. Cellular stress, inflammation, and metabolic dysregulation converge at this point. Novel therapies targeting thermogenic activation and cellular senescence show promise. Despite advancing mechanistic understanding, developing effective interventions remains challenging due to adipose tissue's complex roles in systemic metabolic regulation.

Indexed as

Adipose TissueEnergy MetabolismHomeostasisMetabolic DiseasesAdipocytesAnimalsEndoplasmic Reticulum StressHumansInflammationThermogenesisadipokine dysregulationadipose tissue dysfunctioncellular senescenceinflammatory pathwaysinsulin resistancemetabolic disordersmitochondrial dysfunctionoxidative stress

Identifiers

PMID40630101
PMCPMC12234313

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.