Evidence mapPaperPMID 40640048Full record

ReviewTrends in biochemical sciences2025

The HIF axes in cancer: angiogenesis, metabolism, and immune-modulation.

Karen Acuña-Pilarte, Mei Yee Koh

Abstract readReview
In one paragraph

Review in Trends in biochemical sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed.

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  13. Trefoil Factor-3 Is a Hypoxia-Triggered Pro-Tumorigenic Factor in Hepatoblastoma.Liver international : official journal of the International Association for the Study of the Liver · 2026
    Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Karen Acuña-PilarteDepartment of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA.
Mei Yee KohDepartment of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA. Electronic address: mei.koh@utah.edu.

Funding

A Role for the Novel HAF-NFkappaB Axis in Driving Obesity-Associated Liver CancerR01CA262262 · UTAH STATE HIGHER EDUCATION SYSTEM--UNIVERSITY OF UTAH · 2025 to 2025
$858k
Kidney Cancer Research Program HT9425-24-1-0687NCI NIH HHS R01 CA262262
6 · The paper itself

Abstract

The hypoxia-inducible factors (HIFs) are central transcriptional mediators of the cellular response to hypoxia. HIF activation typically drives a physiologically beneficial adaptive response to hypoxia. However, within solid tumors, the HIF-driven adaptation to hypoxia results in alterations within major cancer cell signaling axes, including those regulating angiogenesis, metabolism, and immune modulation, which profoundly impact tumor progression. This review describes established and recent findings of the role of HIFs in the regulation of these major axes, and the impact of the 'HIF axes' on tumor progression and response to therapy. Current and emerging therapies targeting these axes will also be discussed.

Indexed as

Hypoxia-Inducible Factor 1ImmunomodulationNeoplasmsNeovascularization, PathologicAngiogenesisAnimalsHumansSignal TransductionHypoxia-Inducible Factor 1hypoxiahypoxia-inducible factor (HIF)metabolic reprogrammingsolid tumorstherapeutics targetingtumor microenvironment

Identifiers

PMID40640048
PMCPMC12327054

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.