Evidence mapPaperPMID 40668672Full record

ArticleCell reports2025

Intramuscular adipose tissue restricts functional muscle recovery.

Alessandra M Norris, Victoria R Palzkill, Ambili B Appu, Kiara E Fierman, Christian D Noble, Terence E Ryan, Daniel Kopinke

Abstract read
In one paragraph

Article in Cell reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Article
  5. Article
  6. Review
  7. Article
  8. Article
  9. Review
  10. Fibro-adipogenic progenitor cells from murine SMA muscles are intrinsically adipogenic.Proceedings of the National Academy of Sciences of the United States of America · 2026
    Article
  11. Article
  12. Article
  13. Article
  14. Intramuscular adipose tissue: from progenitor to pathology.American journal of physiology. Cell physiology · 2025
    Review
  15. Ablation of UCP-1+ cells impacts FAP dynamics in muscle regeneration.American journal of physiology. Cell physiology · 2025
    Article
  16. Cellular and molecular bioengineering · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Alessandra M NorrisUniversity of Florida, Department of Pharmacology and Therapeutics, Myology Institute, Gainesville, FL, USA.
Victoria R PalzkillUniversity of Florida, Department of Applied Physiology and Kinesiology, Myology Institute, Gainesville, FL, USA.
Ambili B AppuUniversity of Florida, Department of Pharmacology and Therapeutics, Myology Institute, Gainesville, FL, USA.
Kiara E FiermanUniversity of Florida, Department of Pharmacology and Therapeutics, Myology Institute, Gainesville, FL, USA.
Christian D NobleUniversity of Florida, Department of Pharmacology and Therapeutics, Myology Institute, Gainesville, FL, USA.
Terence E RyanUniversity of Florida, Department of Applied Physiology and Kinesiology, Myology Institute, Gainesville, FL, USA.
Daniel KopinkeUniversity of Florida, Department of Pharmacology and Therapeutics, Myology Institute, Gainesville, FL, USA. Electronic address: dkopinke@ufl.edu.

Funding

Training In Rehabilitation and Neuromuscular PlasticityT32HD043730 · NICHD · UNIVERSITY OF FLORIDA · PI DAVID D FULLER · 2003 to 2026
$5.3M
The role of fatty fibrosis in chronic limb threatening ischemia pathobiologyR01HL171050 · NHLBI · UNIVERSITY OF FLORIDA · PI Daniel Kopinke, Terence E Ryan · 2024 to 2026
$2.0M
Ciliary Hedgehog signaling during adult tissue repair and diseaseR01AR079449 · NIAMS · UNIVERSITY OF FLORIDA · PI Daniel Kopinke · 2022 to 2026
$1.6M
Molecular Mechanisms of Intramuscular Adipose Tissue in Chronic Limb Threating IschemiaF31HL174156 · NHLBI · UNIVERSITY OF FLORIDA · PI PALZKILL, VICTORIA R · 2024 to 2025
$68k
NHLBI NIH HHS F31 HL174156NHLBI NIH HHS R01 HL171050NIAMS NIH HHS R01 AR079449NICHD NIH HHS T32 HD043730
6 · The paper itself

Abstract

With age and disease, skeletal muscle is progressively lost and replaced by fibrotic scar and intramuscular adipose tissue (IMAT). While strongly correlated, it remains unclear whether IMAT has a functional impact on muscle. In the present study, we evaluated the impact of IMAT on muscle regeneration by creating a mouse model where the cellular origin of IMAT, fibro/adipogenic progenitors (FAPs), is prevented from differentiating into adipocytes (mFATBLOCK model). We found that blocking IMAT after an adipogenic injury allowed muscle to regenerate more efficiently, resulting in enhanced functional recovery. Our data explain why acute muscle injuries featuring IMAT infiltration, such as rotator cuff tears and acute denervation injuries, exhibit poor regeneration and lead to a loss of muscle function. It also demonstrates the therapeutic importance of preventing IMAT formation in acute injuries in order to maximize regeneration and minimize loss in muscle mass and function.

Indexed as

Adipose TissueMuscle, SkeletalRecovery of FunctionAdipocytesAdipogenesisAnimalsCell DifferentiationMaleMiceMice, Inbred C57BLRegenerationCP: Metabolismfatty fibrosisfibro/adipogenic progenitorsintramuscular adipose tissuemuscle regenerationrotator cuff tear

Identifiers

PMID40668672
PMCPMC12426892

What Socratic holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.