Evidence mapPaperPMID 40710913Full record

ArticleNeurology international2025

The Association of Axonal Damage Biomarkers and Osteopontin at Diagnosis Could Be Useful in Newly Diagnosed MS Patients.

Eleonora Virgilio, Chiara Puricelli, Nausicaa Clemente, Valentina Ciampana, Ylenia Imperatore, Simona Perga, Sveva Stangalini, Elena Boggio, Alice Appiani, Casimiro Luca Gigliotti and 3 more

Abstract read
In one paragraph

Article in Neurology international, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

13 authors.

Eleonora VirgilioDepartment of Clinical and Biological Sciences, University of Turin, 10100 Turin, Italy.ORCID 0000-0002-0045-3806
Chiara PuricelliClinical Biochemistry, University Hospital Maggiore della Carità di Novara, 28100 Novara, Italy.ORCID 0000-0002-0416-6687
Nausicaa ClementeInterdisciplinary Research Center of Autoimmune Diseases (IRCAD), Department of Health Sciences, University of Piemonte Orientale, Corso Mazzini 18, 28100 Novara, Italy.ORCID 0000-0002-9860-0148
Valentina CiampanaNeurology Unit, Department of Translational Medicine, University of Piemonte Orientale, Maggiore della Carità University-Hospital, 28100 Novara, Italy.
Ylenia ImperatoreNeurology Unit, Department of Translational Medicine, University of Piemonte Orientale, Maggiore della Carità University-Hospital, 28100 Novara, Italy.
Simona PergaClinical Biochemistry, University Hospital Maggiore della Carità di Novara, 28100 Novara, Italy.
Sveva StangaliniClinical Biochemistry, University Hospital Maggiore della Carità di Novara, 28100 Novara, Italy.
Elena BoggioDepartment of Health Sciences, University of Piemonte Orientale, 28100 Novara, Italy.ORCID 0000-0003-2700-3597
Alice AppianiClinical Biochemistry, University Hospital Maggiore della Carità di Novara, 28100 Novara, Italy.
Casimiro Luca GigliottiDepartment of Health Sciences, University of Piemonte Orientale, 28100 Novara, Italy.ORCID 0000-0002-3127-5686
Umberto DianzaniInterdisciplinary Research Center of Autoimmune Diseases (IRCAD), Department of Health Sciences, University of Piemonte Orientale, Corso Mazzini 18, 28100 Novara, Italy.
Cristoforo ComiInterdisciplinary Research Center of Autoimmune Diseases (IRCAD), Department of Health Sciences, University of Piemonte Orientale, Corso Mazzini 18, 28100 Novara, Italy.ORCID 0000-0002-6862-9468
Domizia VecchioInterdisciplinary Research Center of Autoimmune Diseases (IRCAD), Department of Health Sciences, University of Piemonte Orientale, Corso Mazzini 18, 28100 Novara, Italy.ORCID 0000-0001-5920-9210

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

(1) Background: Multiple sclerosis (MS) is a biologically highly heterogeneous disease and has poor predictability at diagnosis. Moreover, robust data indicate that early disease activity strongly correlates with future disability. Therefore, there is a need for strong and reliable biomarkers from diagnosis to characterize and identify patients who require highly effective disease-modifying treatments (DMTs). Several biomarkers are promising, particularly neurofilament light chains (NFLs), but the relevance of others is less consolidated. (2) Methods: We evaluated a panel of axonal damage and inflammatory biomarkers in cerebrospinal fluid (CSF) and matched serum obtained from a cohort of 60 newly diagnosed MS patients. Disability at diagnosis, negative prognostic factors, and the initial DMT prescribed were carefully recorded. (3) Results: We observed correlations between different axonal biomarkers: CSF and serum NFL versus CSF total tau; and between the inflammatory marker osteopontin (OPN) and axonal biomarkers CSF p-Tau, CSF total tau, and serum NFL. CSF and serum NFL and total tau, as well as CSF OPN, positively correlated with EDSS at diagnosis. Moreover, CSF and serum NFL levels were increased in patients with gadolinium-enhancing lesions (

Indexed as

biomarkermultiple sclerosisneurodegenerationneurofilamentosteopontintau

Identifiers

PMID40710913
PMCPMC12300847

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.