Evidence map›Paper›PMID 40720255›Full record

ReviewPhysiological reviews2026

Metabolic adaptations in cancer progression.

Yiming Peng-Winkler, Sarah-Maria Fendt

Abstract readReview
In one paragraph

Review in Physiological reviews, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed.

  1. Hyperpolarized [2-NMR in biomedicine · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Yiming Peng-WinklerLaboratory of Cellular Metabolism and Metabolic Regulation, VIB-KU Leuven Center for Cancer Biology, Leuven, Belgium.ORCID 0000-0002-8327-559X
Sarah-Maria FendtLaboratory of Cellular Metabolism and Metabolic Regulation, VIB-KU Leuven Center for Cancer Biology, Leuven, Belgium.ORCID 0000-0001-6018-9296

Funding

Beug FoundationDeutsche Forschungsgemeinschaft (DFG) 521676159Fondation Francqui - Stichting (La Fondation Francqui)Fonds Baillet LatourFonds Wetenschappelijk Onderzoek (FWO)Foundation ARCiBOF (INTERCEPt)KU Leuven (Katholieke Universiteit Leuven)Stichting Tegen Kanker (Fondation Contre le Cancer)Wereld Kanker Onderzoek Fonds (WKOF)World Cancer Research Fund International (WCRF) IIG_FULL_2023_001
6 · The paper itself

Abstract

Cancer cells reprogram their metabolism as they travel to distant organs to establish metastases, the leading cause of cancer-related mortality. Although the metabolic state of primary tumors has been extensively studied, the specific metabolic alterations associated with metastases have only recently garnered significant attention. The metabolic dependencies that arise during the metastatic cascade, along with the adaptive metabolic shifts required for growth in a new microenvironment, present promising therapeutic targets. In this review, we provide an overview of cancer metabolism, followed by a detailed exploration of the metabolic changes occurring at each stage of metastasis and within common organs of metastatic spread. Finally, we examine the potential and challenges of targeting metabolic pathways in cancer therapy.

Indexed as

Adaptation, PhysiologicalEnergy MetabolismNeoplasmsAnimalsDisease ProgressionHumansNeoplasm MetastasisTumor Microenvironmentcancermetabolismmetabolism-based therapymetastasisorgan microenvironment

Identifiers

PMID40720255
PMCPMC7618854

What Socratic holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.