Evidence mapPaperPMID 40725403Full record

ArticleGenes2025

Hwanhon Decoction Ameliorates Cognitive Impairment and Suppresses Neuroinflammation in a Chronic Cerebral Hypoperfusion Mouse Model: Involvement of Key Genes Identified by Network Pharmacology.

Sieun Kang, Chiyeon Lim, Sehyun Lim, Kyoung-Min Kim, Suin Cho

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In one paragraph

Article in Genes, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Sieun KangDepartment of Korean Medicine, College of Korean Medicine, Dong-Eui University, Busan 47227, Republic of Korea.
Chiyeon LimDepartment of Radiology, Massachusetts General Hospital and Harvard Medical School, Charlestown, MA 02129, USA.ORCID 0000-0003-0178-6976
Sehyun LimDepartment of Radiology, Massachusetts General Hospital and Harvard Medical School, Charlestown, MA 02129, USA.
Kyoung-Min KimDepartment of Korean Medicine, College of Korean Medicine, Dong-Eui University, Busan 47227, Republic of Korea.
Suin ChoDepartment of Korean Medicine, School of Korean Medicine, Pusan National University, Yangsan 50612, Republic of Korea.ORCID 0000-0001-8507-1382

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundWith an aging population, dementia prevalence is increasing in Korea. Vascular dementia (VaD), often caused by cerebrovascular disease (CVD), is more common in Korea compared to Western countries. Hwanhon decoction, a traditional medicine containing Ephedrae Herba, Armeniacae Semen, and Glycyrrhizae Radix et Rhizoma, is traditionally used for CVD-related loss of consciousness. This study aimed to assess the cognitive improvement and anti-inflammatory effects of Hwanhon decoction extract (HHex) in a mouse model of VaD caused by chronic cerebral hypoperfusion (CCH).

methodsKey pharmacologically active ingredients of Hwanhon decoction were identified using network pharmacology analysis. VaD was induced in C57Bl/6 male mice through bilateral common carotid artery stenosis (BCAS). Mice were divided into sham surgery, BCAS control, low-dose HHex (L-HHex), and high-dose HHex (H-HHex) groups (

resultsHHex administration reduced cognitive impairment and mitigated CCH-induced astrocyte activation. Inflammatory responses mediated by reactive astrocytes were suppressed, and network pharmacology predicted central proteins influencing HHex's activity.

conclusionsHHex alleviated cognitive dysfunction and reduced inflammation in a VaD mouse model, suggesting its potential as a therapeutic agent for vascular dementia associated with impaired cerebral blood flow.

Indexed as

Cognitive DysfunctionDementia, VascularDrugs, Chinese HerbalNeuroinflammatory DiseasesAnimalsDisease Models, AnimalMaleMiceMice, Inbred C57BLNetwork PharmacologyDrugs, Chinese Herbalbilateral common carotid artery stenosischronic cerebral hypoperfusioncognitive impairmentneuroinflammationvascular dementia

Identifiers

PMID40725403
PMCPMC12295633

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.