Evidence mapPaperPMID 40755151Full record

ArticleActa obstetricia et gynecologica Scandinavica2025

Association of maternal asthma with congenital heart defects in offspring.

Mari Kemppainen, Turkka Kirjavainen, Emmi Helle, Mika Gissler

Abstract read
In one paragraph

Article in Acta obstetricia et gynecologica Scandinavica, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Association of maternal asthma with congenital heart defects in offspring.Acta obstetricia et gynecologica Scandinavica · 2025
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Mari KemppainenVantaa and Kerava Wellbeing County, Martinlaakso Health Station, Vantaa, Finland.ORCID 0000-0002-5680-7394
Turkka KirjavainenDepartment of Pediatrics, New Children's Hospital, Helsinki, Finland.ORCID 0000-0002-0043-5093
Emmi HelleStem Cells and Metabolism Research Program, RPU, University of Helsinki, Helsinki, Finland.ORCID 0000-0001-8993-2194
Mika GisslerDepartment of Data and Analytics, Finnish Institute for Health and Welfare, Helsinki, Finland.ORCID 0000-0001-8254-7525

Funding

European Research Council 101076986Foundation for Pediatric Research in Finland 190140Foundation for Pediatric Research in Finland 200184Foundation for Pediatric Research in Finland 220135State research fundTampere Tuberkuloosisäätiö fund
6 · The paper itself

Abstract

introductionAsthma is the most common chronic disease among pregnant women. Some studies have linked maternal asthma to an increased risk of congenital malformations, particularly congenital heart defects (CHDs). Among these, ventricular septal defect (VSD) is the most common, accounting for nearly half of all CHD cases. We investigated the association between maternal asthma and the risk of CHD in offspring. As a sensitivity analysis, we also examined the associations between maternal atopic predisposition, maternal use of asthma medication, and CHD risk. MATERIAL AND

methodsThis retrospective, population-based study utilized a comprehensive birth cohort of all singleton births in Finland between 1996 and 2018. Data were derived by linking four high-coverage national health registers.

resultsThe cohort included 1 274 540 eligible mother-child pairs. A total of 106 544 (8.4%) mothers had a diagnosis of asthma or had purchased asthma medication during pregnancy. Of the offspring, 18 772 (1.5%) were diagnosed with a congenital heart defect (CHD), and 8855 (0.7%) had a diagnosis of ventricular septal defect (VSD). Maternal asthma was weakly associated with overall CHD (OR 1.18; 95% CI, 1.12 to 1.24) and with VSD (OR 1.19; 95% CI, 1.10 to 1.27). The association between maternal asthma and CHD was observed in both allergic asthma (OR 1.33; 95% CI, 1.18 to 1.50) and nonallergic asthma (OR 1.27; 95% CI, 1.04 to 1.56). Maternal allergic conditions, such as atopic dermatitis and allergic rhinitis, were also associated with CHD overall (OR 1.13; 95% CI, 1.02 to 1.25) and with VSD (OR 1.20; 95% CI, 1.04 to 1.38).

conclusionsMaternal asthma and allergic tendency are associated with a modestly increased risk of CHD in offspring, particularly VSD. Nonallergic asthma was also linked to CHD, especially right-sided defects. These findings suggest that maternal allergic inflammation may have teratogenic effects during fetal cardiac development.

Indexed as

AsthmaHeart Defects, CongenitalPregnancy ComplicationsPrenatal Exposure Delayed EffectsAdultFemaleFinlandHeart Septal Defects, VentricularHumansInfant, NewbornMalePregnancyRetrospective StudiesRisk Factorsallergyasthmacongenital heart defectheart septal defects

Identifiers

PMID40755151
PMCPMC12451212

What Socratic holds

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LicenceCC BY-NC
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.