Evidence mapPaperPMID 40764579Full record

ArticleBMC medicine2025

Insulin resistance as a potential driving force of parental obesity-induced adverse metabolic programming mechanisms in children with obesity.

Lucía Jurado-Sumariva, Álvaro González-Domínguez, Otto Savolainen, Jesús Domínguez-Riscart, Rikard Landberg, Raúl González-Domínguez

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In one paragraph

Article in BMC medicine, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Article
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  3. Review
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Lucía Jurado-SumarivaInstituto de Investigación e Innovación Biomédica de Cádiz (INiBICA), Hospital Universitario Puerta del Mar, Universidad de Cádiz, Cádiz, 11009, Spain.
Álvaro González-DomínguezInstituto de Investigación e Innovación Biomédica de Cádiz (INiBICA), Hospital Universitario Puerta del Mar, Universidad de Cádiz, Cádiz, 11009, Spain. alvaro.gonzalez@inibica.es.
Otto SavolainenDivision of Food and Nutrition Science, Department of Life Sciences, Chalmers University of Technology, Gothenburg, SE-412 96, Sweden.
Jesús Domínguez-RiscartInstituto de Investigación e Innovación Biomédica de Cádiz (INiBICA), Hospital Universitario Puerta del Mar, Universidad de Cádiz, Cádiz, 11009, Spain.
Rikard LandbergDivision of Food and Nutrition Science, Department of Life Sciences, Chalmers University of Technology, Gothenburg, SE-412 96, Sweden.
Raúl González-DomínguezInstituto de Investigación e Innovación Biomédica de Cádiz (INiBICA), Hospital Universitario Puerta del Mar, Universidad de Cádiz, Cádiz, 11009, Spain. raul.gonzalez@inibica.es.

Funding

EMBO 9400Fundación para la Gestión de la Investigación Biomédica de Cádiz LII19/16IN-CO24Instituto de Salud Carlos III CM23/00026Instituto de Salud Carlos III PI22/01899Ministerio de Educación y F CAS22/00080
6 · The paper itself

Abstract

backgroundParental obesity has been identified as one of the most important early risk factors for childhood obesity, but molecular mechanisms driving this greater predisposition remain to be elucidated.

methodsIn this study, we recruited a cohort comprising children with obesity (body mass index over two z-scores above the age/sex-adjusted mean of the Spanish reference population, age range: 6-12 years), born to parents with obesity (N = 18) or without obesity (N = 41), as well as matched healthy controls (N = 26). Plasma and erythrocyte samples were collected for comprehensive biochemical and metabolomics analyses, this latter by applying high-throughput liquid chromatography-mass spectrometry. Then, a combination of multivariate and univariate statistical tools was applied to unravel the molecular pathogenic impairments that parental obesity may imprint in the offspring.

resultsInterestingly, we found parental obesity to be associated with exacerbated unhealthy metabolic outcomes in the offspring with obesity, as mirrored in higher fasting insulin levels (p = 2.8 × 10

conclusionsTherefore, we hypothesize that insulin resistance could be a major driving force in mediating deleterious programming mechanisms induced by parental obesity in the offspring.

Indexed as

Insulin ResistanceObesityPediatric ObesityBody Mass IndexCase-Control StudiesChildFemaleHumansMaleMetabolic ReprogrammingMetabolomicsParentsChildhood obesityInsulin resistanceMetabolomicsParental obesity

Identifiers

PMID40764579
PMCPMC12326771

What Socratic holds

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LicenceCC BY-NC-ND
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.