ReviewHead & face medicine2025
The effects of mitochondrial damage and energy metabolism disorders on the progression of noise-induced deafness and its treatment.
Review in Head & face medicine, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
2 citing papers in PubMed.
- Association between combined occupational exposure to coal dust and noise and high-frequency hearing loss among workers: a cross-sectional study.Frontiers in public health · 2026Article
- Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
Abstract
Noise-induced hearing loss (NIHL) is a major cause of hearing impairment globally, affecting numerous individuals. The pathogenesis of NIHL is complex, involving multiple mechanisms such as mechanical damage, oxidative stress, mitochondrial dysfunction, and energy metabolism disorders. Although these mechanisms are implicated, the complete understanding of NIHL's pathogenesis remains elusive. In recent years, the role of mitochondrial dysfunction and energy metabolism disorders in NIHL has garnered significant attention. This review aims to provide a comprehensive summary of the pathological mechanisms underlying NIHL, focusing on the contributions of mitochondrial damage and energy metabolism disorders. Moreover, this review evaluates existing therapeutic approaches and proposes future research directions through critical analysis of published evidence.
Indexed as
Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.