Evidence mapPaperPMID 40778010Full record

ReviewThe Journal of clinical and aesthetic dermatology2025

Structural Insights: What Makes Some PDE4 Inhibitors More Effective in Inflammatory Dermatoses.

Naiem T Issa, Jimin Wang, Ailish Hanly, Minh Ho, Sabine Obagi, Giovanni Damiani, James Q Del Rosso, Youna Kang, Christopher G Bunick

Abstract readReview
In one paragraph

Review in The Journal of clinical and aesthetic dermatology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Naiem T IssaDr. Issa is with Forefront Dermatology in Vienna, Virginia; the University of Miami Miller School of Medicine in Miami, Florida; the Dr. Phillip Frost Department of Dermatology and Cutaneous Surgery in Miami, Florida; and the Department of Dermatology at George Washington University School of Medicine and Health Sciences in Washington, District of Columbia.
Jimin WangDrs. Wang and Bunick are with the Department of Molecular Biophysics and Biochemistry at Yale University in New Haven, Connecticut.
Ailish HanlyDrs. Bunick, Kang, Hanly, and Mr. Ho are with the Department of Dermatology at Yale School of Medicine in New Haven, Connecticut.
Minh HoDrs. Bunick, Kang, Hanly, and Mr. Ho are with the Department of Dermatology at Yale School of Medicine in New Haven, Connecticut.
Sabine ObagiMs. Obagi is with the College of Medicine at the University of Arizona in Tucson, Arizona.
Giovanni DamianiDr. Damiani is with the Department of Biomedical, Surgical, and Dental Sciences at the University of Milan in Milan, Italy; and Fondazione IRCCS CA' Granda Ospedale Maggiore Policlinico in Milan, Italy.
James Q Del RossoDr. Del Rosso is with JDR Dermatology Research in Las Vegas, Nevada; Advanced Dermatology and Cosmetic Surgery in Maitland, Florida; and the Department of Dermatology at Touro University Nevada in Henderson, Nevada.
Youna KangDrs. Bunick, Kang, Hanly, and Mr. Ho are with the Department of Dermatology at Yale School of Medicine in New Haven, Connecticut.
Christopher G BunickDrs. Wang and Bunick are with the Department of Molecular Biophysics and Biochemistry at Yale University in New Haven, Connecticut.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Psoriasis, seborrheic dermatitis, and atopic dermatitis are chronic inflammatory skin diseases affecting millions of people in the United States and worldwide across the human lifespan. The immune pathways underlying the pathogenesis of these dermatoses include Type I (IFN-γ, TNF-α), Type II (IL-4, IL-5, IL-13), and Type III (IL- 17A/F, IL-22, IL-23) cytokines. These cytokines function downstream of the enzyme phosphodiesterase-IV (PDE4), which makes PDE4 an upstream central regulator of inflammatory dermatoses. PDE4, therefore, is a key drug target for alleviating inflammatory skin diseases. In this brief review, we discuss and simplify into clinically relevant terminology the molecular findings of a 2024 study by Wang et al, which analyzed the structural properties of dermatologic PDE4 inhibitors and thereby provided molecular rationale as to why roflumilast has the greatest potency and is highly efficacious across multiple inflammatory dermatoses.

Indexed as

apremilastatopic dermatitiscAMPcrisaborolePsoriasisroflumilastseborrheic dermatitis

Identifiers

PMID40778010
PMCPMC12327561

What Socratic holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.