Evidence map›Paper›PMID 40778304›Full record

ReviewFrontiers in aging neuroscience2025

NMDA receptors in neurodegenerative diseases: mechanisms and emerging therapeutic strategies.

Keyi Zhang, Ming Wen, Xinyue Nan, Shuaizhu Zhao, Hao Li, Yanping Ai, Houze Zhu

Abstract readReview
In one paragraph

Review in Frontiers in aging neuroscience, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed.

  1. Review
  2. Phytochemicals with potential NMDAR-inhibitory activities: a review of neuroprotective mechanisms against excitotoxicity.Saudi pharmaceutical journal : SPJ : the official publication of the Saudi Pharmaceutical Society · 2026
    Review
  3. Article
  4. Reduced expression of N-methyl-D-aspartate receptor and calcium signaling genes in gray matter is associated with cancer-related cognitive impairment.Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer · 2026
    Article
  5. Review
  6. Review
  7. Article
  8. Neurotransmitter Systems in Alzheimer's Disease.Current issues in molecular biology · 2026
    Review
  9. Article
  10. Article
  11. Article
  12. Article
  13. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Keyi Zhang *Innovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Ming Wen *Innovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Xinyue NanInnovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Shuaizhu ZhaoInnovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Hao LiInnovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Yanping AiInnovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.
Houze ZhuInnovation Center for Brain Medical Sciences, The Ministry of Education of the People's Republic of China, Huazhong University of Science and Technology, Wuhan, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

NMDA receptors (NMDARs) are widely distributed throughout the central nervous system (CNS) and play pivotal roles in normal physiological processes such as synaptic plasticity, learning, and memory. Substantial evidence indicates that NMDAR dysfunction, particularly excessive calcium influx, critically contributes to the pathogenesis of major neurodegenerative diseases, including Alzheimer's disease (AD), Parkinson's disease (PD), Huntington's disease (HD), and amyotrophic lateral sclerosis (ALS). Dysregulated glutamatergic signaling synergizes with pathological protein aggregation (e.g., Aβ,

Indexed as

Alzheimer’s disease (AD)amyotrophic lateral sclerosis (ALS)excitotoxicityglutamateHuntington’s disease (HD)neurodegenerative diseaseNMDA receptors (N-methyl-D-aspartate receptors)Parkinson’s disease (PD)

Identifiers

PMID40778304
PMCPMC12328396

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.