Evidence map›Paper›PMID 40797279›Full record

ReviewJournal of hematology & oncology2025

Target neutrophil heterogeneity and plasticity in cancer.

Ye Feng, Guang Liu, Haiqing Li, Lin Cheng

Abstract readReview
In one paragraph

Review in Journal of hematology & oncology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 21 papers.

0numbers the graph read from it
0cells of the map it votes in
21citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

21 citing papers in PubMed.

  1. Review
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  11. Metabolic Control of Immunity-Unveiling Neutrophil Mechanisms.Advances in experimental medicine and biology · 2026
    Review
  12. Review
  13. Review
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  17. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Ye FengShanghai Sunshine Rehabilitation Center (Shanghai YangZhi Rehabilitation Hospital), Tongji University School of Medicine, Shanghai, 201619, China. fengye0509@163.com.
Guang LiuDepartment of Vascular Surgery, Shanghai Ninth People's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200023, China. liug112050@sh9hospital.org.cn.
Haiqing LiDepartment of Cardiac Surgery, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China. drlihaiqing@163.com.
Lin ChengShanghai Institute of Hematology, State Key Laboratory of Medical Genomics, National Research Center for Translational Medicine at Shanghai, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200025, China. lincheng@shsmu.edu.cn.

Funding

National clinical key specialty construction project of China Z155080000004National Key R&D Program of China 2024YFA1803503Rehabilitation Policy Research Project of Shanghai Disabled Persons' Federation 2023 ZC1014Research Project of China Disabled Person's Federation - on assistive technology 2023CDPFAT-30Shanghai Clinical Research Ward SHDC2023CRW018BShanghai Hospital Development Center Foundation-Shanghai Municipal Hospital Rehabilitation Medicine Specialty Alliance SHDC22023304Shanghai Municipal Education Commission 22SG12Shanghai Municipal Health Commission 2022XD050Shanghai Research Center of Rehabilitation Medicine (Top Priority Research Center of Shanghai) 2023ZZ02027Talent Cultivation Plan of Shanghai YangZhi Rehabilitation Hospital (Shanghai Sunshine Rehabilitation Center) 2022GGRC06
6 · The paper itself

Abstract

Neutrophils have long been regarded as cells of a limited lifespan, known to produce pro-inflammatory molecules, and primarily engaged in combating infections. However, recent advancements in single-cell analysis and molecular biology have revealed their remarkable heterogeneity and plasticity, particularly within the context of tumors. This review explores the development and diversity of neutrophils under both physiological and pathological conditions, with a particular focus on their roles in cancer. The discussion encompasses the emergence of distinct neutrophil subtypes, particularly senescent neutrophils, within tumors and their context-dependent functions in tumorigenesis, progression, metastasis, and recurrence. The plasticity of these cells, driven by intrinsic factors and the tumor microenvironment, allows them to be reprogrammed between pro-tumor and anti-tumor phenotypes. This process is influenced by cytokines, metabolic reprogramming, and interactions with other immune cells. The potential of targeting and engineering neutrophil as a therapeutic avenue for cancer treatment is further underscored, including the use of senolytic agents, metabolic inhibitors, and reprogramming strategies. Finally, future research directions are proposed to further elucidate the mechanisms underlying neutrophil heterogeneity and plasticity, with the aim of developing novel therapeutic approaches to modulate neutrophil function in cancer.

Indexed as

Cell PlasticityNeoplasmsNeutrophilsAnimalsHumansTumor MicroenvironmentCancerHeterogeneityNeutrophilPlasticityTargeting

Identifiers

PMID40797279
PMCPMC12345094

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.