Evidence mapPaperPMID 40800604Full record

ArticleBiochemistry and biophysics reports2025

Computational identification of key genetic drivers in COPD: A first step towards uncovering candidate biomarkers in smokers.

Arman Mokaram Doust Delkhah, Ali Ghazvini, Masoud Arabfard

Abstract read
In one paragraph

Article in Biochemistry and biophysics reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Arman Mokaram Doust DelkhahChemical Injuries Research Center, Systems Biology and Poisonings Institute, Baqiyatallah University of Medical Sciences, Tehran, Iran.
Ali GhazviniChemical Injuries Research Center, Systems Biology and Poisonings Institute, Baqiyatallah University of Medical Sciences, Tehran, Iran.
Masoud ArabfardChemical Injuries Research Center, Systems Biology and Poisonings Institute, Baqiyatallah University of Medical Sciences, Tehran, Iran.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Chronic obstructive pulmonary disease (COPD) is a leading challenge of global public health that predominantly affects developing countries. Although smoking is the main risk factor, only a fraction of smokers develop COPD. This study aimed to identify biomarkers or therapeutic targets that would effectively aid early diagnosis and treatment of smoking-induced COPD. Methods and results: After retrieving GSE27597, GSE38974, GSE47460, GSE76925, and GSE239897 from the Gene Expression Omnibus, never-smokers were excluded from each dataset. Differential expression analysis and weighted gene co-expression network analysis (WGCNA) were employed to discern a reliable gene list. Subsequently, integrated data, which incorporated 120 control and 349 COPD samples, was analyzed by random forest (RF) and least absolute shrinkage and selection operator (LASSO) methods to identify key genes. Lastly, 6 genes with the area under the receiver operating characteristic curve exceeding 0.7 were selected as potential biomarkers of smoking-induced COPD. Conclusion: These results suggested

Indexed as

BiomarkerCigarette smokingCOPDGene expression analysisMachine learning

Identifiers

PMID40800604
PMCPMC12340515

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.