Evidence mapPaperPMID 40869281Full record

ReviewInternational journal of molecular sciences2025

The Multifaceted Role of Mitochondria in Angiogenesis.

Sara Cannito, Ida Giardino, Maria d'Apolito, Massimo Pettoello-Mantovani, Francesca Scaltrito, Domenica Mangieri, Annamaria Piscazzi

Abstract readReview
In one paragraph

Review in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed.

  1. Review
  2. Article
  3. [Research progress on the metabolic regulatory mechanisms of coronary collateral circulation].Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences · 2026
    Review
  4. Article
  5. Review
  6. Review
  7. Review
  8. Review
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Sara CannitoLaboratory Medicine, Department of Clinical and Experimental Medicine, University of Foggia, 71122 Foggia, Italy.
Ida GiardinoLaboratory Medicine, Department of Clinical and Experimental Medicine, University of Foggia, 71122 Foggia, Italy.
Maria d'ApolitoMedical Genetics, Department of Clinical and Experimental Medicine, University of Foggia, 71122 Foggia, Italy.ORCID 0000-0001-9170-4668
Massimo Pettoello-MantovaniItalian Academy of Pediatrics, 20126 Milan, Italy.
Francesca ScaltritoResidency Course in Pediatrics, Department of Medical and Surgical Sciences, University of Foggia, 71122 Foggia, Italy.
Domenica MangieriApplied Biology, Department of Clinical and Experimental Medicine, University of Foggia, 71122 Foggia, Italy.ORCID 0000-0001-7350-9383
Annamaria PiscazziLaboratory Medicine, Department of Clinical and Experimental Medicine, University of Foggia, 71122 Foggia, Italy.ORCID 0000-0003-2772-3869

Funding

European Union-Next Generation EU through the Italian Ministry of University and Research under PNRR-M4C2- I1.3 Project PE_00000019 "HEAL ITALIA" to Lorenzo Lo Muzio CUP UNIFG D73C22001230006 PNRR-M4C2- I1.3 Project PE_00000019 CUP UNIFG D73C22001230006
6 · The paper itself

Abstract

Angiogenesis, the formation of new blood vessels from pre-existing ones, is crucial for various physiological and pathological conditions, including embryonic development, wound healing, tissue regeneration and tumor progression. While traditionally attributed to the actions of growth factors and their receptors, emerging evidence highlights the crucial regulatory roles of mitochondria in angiogenesis. This narrative review explores the multifaceted functions of mitochondria in endothelial cells, which are central to blood vessel formation. Beyond their classical role in ATP production, mitochondria contribute to angiogenesis through redox signaling, calcium homeostasis, biosynthetic activity, and reactive oxygen species (ROS) generation. These organelles help regulate key endothelial behaviors such as proliferation, migration, and tube formation through mechanisms that include mitochondrial calcium signaling and ROS-mediated stabilization of hypoxia-inducible factor-1α (HIF-1α), leading to increased vascular endothelial growth factor (VEGF) expression. Additionally, mitochondrial dynamics, dysfunction, and genetic factors are discussed for their influence on angiogenic outcomes. Understanding these complex mitochondrial functions opens new therapeutic avenues for modulating angiogenesis in diseases such as cancer and cardiovascular disorders.

Indexed as

MitochondriaNeovascularization, PathologicNeovascularization, PhysiologicAngiogenesisAnimalsEndothelial CellsHumansHypoxia-Inducible Factor 1, alpha SubunitReactive Oxygen SpeciesSignal TransductionVascular Endothelial Growth Factor AHypoxia-Inducible Factor 1, alpha SubunitReactive Oxygen SpeciesVascular Endothelial Growth Factor Aangiogenesismitochondriamitochondrial calcium transportmtROS-HIF1a-VEGF axisVEGFR2

Identifiers

PMID40869281
PMCPMC12386899

What Socratic holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.