Evidence mapPaperPMID 40906143Full record

ArticleBiology2025

Exercise Ameliorates Dopaminergic Neurodegeneration in Parkinson's Disease Mice by Suppressing Microglia-Regulated Neuroinflammation Through Irisin/AMPK/Sirt1 Pathway.

Bin Wang, Nan Li, Yuanxin Wang, Xin Tian, Junjie Lin, Xin Zhang, Haocheng Xu, Yu Sun, Renqing Zhao

Abstract read
In one paragraph

Article in Biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Brain-muscle axis regulation of neuroinflammation and sarcopenia in Parkinson's disease: the bridging role of lactylation.Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology · 2026
    Review
  4. Neurobiology of exercise in Parkinson's disease.Journal of Parkinson's disease · 2026
    Review
  5. Article
  6. Review
  7. Review
  8. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Bin WangCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.ORCID 0009-0000-0066-8084
Nan LiCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.
Yuanxin WangCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.
Xin TianCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.
Junjie LinCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.ORCID 0000-0002-2252-0985
Xin ZhangCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.
Haocheng XuCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.ORCID 0000-0003-2369-305X
Yu SunCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.
Renqing ZhaoCollege of Physical Education, Yangzhou University, Yangzhou 225009, China.ORCID 0000-0001-8901-9506

Funding

Natural Science Foundation of Jiangsu Province BK20201435
6 · The paper itself

Abstract

Although exercise is known to exert anti-inflammatory effects in neurodegenerative diseases, its specific impact and underlying mechanisms in Parkinson's disease (PD) remain poorly understood. This study explores the effects of exercise on microglia-mediated neuroinflammation and apoptosis in a PD model, focusing on the role of irisin signaling in mediating these effects. Using a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced PD mouse model, we found that a 10-week treadmill exercise regimen significantly enhanced motor function, reduced dopaminergic neuron loss, attenuated neuronal apoptosis, and alleviated neuroinflammation. Exercise also shifted microglia from a pro-inflammatory to an anti-inflammatory phenotype. Notably, levels of irisin, phosphorylated AMP-activated protein kinase (p-AMPK), and sirtuin 1 (Sirt1), which were decreased in the PD brain, were significantly increased following exercise. These beneficial effects were abolished by blocking the irisin receptor with cyclic arginine-glycine-aspartic acid-tyrosine-lysine (cycloRGDyk). Our results indicate that exercise promotes neuroprotection in PD by modulating microglial activation and the AMPK/Sirt1 pathway through irisin signaling, offering new insights into exercise-based therapeutic approaches for PD.

Indexed as

apoptosisexerciseirisinmicroglianeuroinflammationParkinson’s Disease

Identifiers

PMID40906143
PMCPMC12383941

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.