Evidence mapPaperPMID 40919715Full record

ReviewEndocrine-related cancer2025

Defining aging-associated factors that increase susceptibility to prostate cancer.

Shile Zhang, Ernie Lee, Sachi Bopardikar, Andrew S Goldstein

Abstract readReview
In one paragraph

Review in Endocrine-related cancer, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Shile ZhangDepartment of Molecular and Medical Pharmacology, UCLA David Geffen School of Medicine, Los Angeles, California, USA.
Ernie LeeDepartment of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, California, USA.
Sachi BopardikarDepartment of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, California, USA.
Andrew S GoldsteinDepartment of Molecular, Cell and Developmental Biology, University of California Los Angeles, Los Angeles, California, USA.ORCID https://orcid.org/0000-0003-0434-9149

Funding

UCLA Clinical and Translational Science InstituteUL1TR001881 · UNIVERSITY OF CALIFORNIA LOS ANGELES · 2025 to 2025
$9.9M
UCLA SPORE IN PROSTATE CANCERP50CA092131 · UNIVERSITY OF CALIFORNIA LOS ANGELES · 2002 to 2005
$8.7M
NCATS NIH HHS UL1 TR001881NCI NIH HHS P50 CA092131NCI NIH HHS R01 CA237191
6 · The paper itself

Abstract

Age is a major risk factor for a range of diseases, including prostate cancer. Understanding how age influences the susceptibility of normal prostate epithelial cells to cancer initiation is complicated by the fact that aging affects all tissues in the body. Assessing how various aging mechanisms influence the prostate epithelium is a necessary step to determine the critical factors associated with aging that increase prostate cancer risk. Here, we take a broad view of both prostate epithelial cell-intrinsic and cell-extrinsic factors that change with age and are likely to contribute to age-related risk of prostate tumorigenesis. For each factor, we discuss the impact of these age-related changes on cancer risk. We highlight important areas where additional research is required to help decipher the specific age-associated changes that contribute to prostate cancer initiation. Finally, we address the potential impact of various therapeutic interventions on aging phenotypes and cancer risk.

Indexed as

AgingProstatic NeoplasmsAnimalsDisease SusceptibilityHumansMaleRisk Factorsagingepitheliumprostate

Identifiers

PMID40919715
PMCPMC12476893

What Socratic holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.