Evidence mapPaperPMID 40942007Full record

ReviewMolecules (Basel, Switzerland)2025

Biologics as Therapeutical Agents Under Perspective Clinical Studies for Alzheimer's Disease.

Huan Li, Xinai Shen, Beiyu Zhang, Zheying Zhu

Abstract readReview
In one paragraph

Review in Molecules (Basel, Switzerland), 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. A randomized phase 1b/2 trial of ABBV-916 in adults with early Alzheimer's disease.Alzheimer's & dementia : the journal of the Alzheimer's Association · 2026
    Trial
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Huan LiDivision of Molecular Therapeutics and Formulation, School of Pharmacy, The University of Nottingham, Nottingham NG7 2RD, UK.ORCID 0009-0002-7090-2221
Xinai ShenDivision of Molecular Therapeutics and Formulation, School of Pharmacy, The University of Nottingham, Nottingham NG7 2RD, UK.ORCID 0009-0007-2112-0510
Beiyu ZhangDivision of Molecular Therapeutics and Formulation, School of Pharmacy, The University of Nottingham, Nottingham NG7 2RD, UK.
Zheying ZhuDivision of Molecular Therapeutics and Formulation, School of Pharmacy, The University of Nottingham, Nottingham NG7 2RD, UK.ORCID 0000-0002-2135-3812

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Alzheimer's disease (AD) is a progressive neurodegenerative disorder characterised by cognitive decline, synaptic loss, and multifaceted pathology involving amyloid-β (Aβ) aggregation, tau hyperphosphorylation, neuroinflammation, and impaired proteostasis. In recent years, biologic therapies, such as monoclonal antibodies, vaccines, antisense oligonucleotides (ASOs), and gene therapies, have gained prominence as promising disease-modifying strategies. In this review, we provide a comprehensive synthesis of current biologic approaches under clinical evaluation for AD. Drawing on data curated from

Indexed as

Alzheimer DiseaseBiological ProductsAmyloid beta-PeptidesAnimalsClinical Trials as TopicGenetic TherapyHumansOligonucleotides, Antisensetau ProteinsAmyloid beta-PeptidesBiological ProductsOligonucleotides, Antisensetau Proteinsdisease-modifying therapiesgene therapyimmunotherapyneurodegenerative diseaseneuroinflammationRNA therapeutics

Identifiers

PMID40942007
PMCPMC12429904

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.