Evidence mapPaperPMID 40949582Full record

ArticleJournal of medical signals and sensors2025

Investigating Causal Links between Metabolite Profiles and Ulcerative Colitis: A Bidirectional Mendelian Randomization Study.

Parvin Zarei, Zoha Kamali, Ammar Hassanzadeh Keshteli, Peyman Adibi Sedeh, Ahmad Vaez

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Article in Journal of medical signals and sensors, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

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2 · The registry

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3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Parvin ZareiDepartment of Bioinformatics, Isfahan University of Medical Sciences, Isfahan, Iran.
Zoha KamaliDepartment of Bioinformatics, Isfahan University of Medical Sciences, Isfahan, Iran.
Ammar Hassanzadeh KeshteliDepartment of Medicine, University of Alberta, Edmonton, Canada.
Peyman Adibi SedehIsfahan Gastroenterology and Hepatology Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Ahmad VaezDepartment of Bioinformatics, Isfahan University of Medical Sciences, Isfahan, Iran.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: While metabolic biomarkers are known to play a significant role in the development of ulcerative colitis (UC), the exact causal relationships between them remain uncertain and warrant further investigations. Here we report a bidirectional two-sample Mendelian randomization (MR) study to evaluate causal relationships between 503 blood metabolites and UC. Methods: We used genome-wide association study (GWAS) data on blood metabolite levels from two separate studies on European individuals ( Results: Our study suggests that total lipids in small high-density lipoprotein levels (S.HDL.L) are marginal significant positive associated with the development of UC (odds ratio = 1.167, 95% confidence interval: 0.998-1.364, Conclusions: Total lipids in S.HDL.L may offer a potential trend as valuable circulating metabolic biomarkers for the screening and prevention of UC in clinical practice. In addition, they could serve as potential candidate molecules for elucidating the mechanisms underlying UC and for identifying suitable drug targets.

Indexed as

Biomarkerscausalitycolitisinflammatory bowel diseasesulcerative

Identifiers

PMID40949582
PMCPMC12431707

What Socratic holds

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.