ReviewAdvances in experimental medicine and biology2025
Apoptosis and Cell Clearance in Skin Wound Healing.
Review in Advances in experimental medicine and biology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Multifunctional Prussian-Blue-Based Hydrogel for Photothermal Antibacterial and Infected Wound Regeneration.Polymers · 2026Article
- Total Saponins from Rhizoma Panacis Majoris Promote Wound Healing in Diabetic Rats by Regulating Inflammatory Dysregulation.International journal of molecular sciences · 2026Article
- An Update of Phytotherapeutic Advances of Marigold (Plants (Basel, Switzerland) · 2025Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The skin is our body's largest natural barrier, and daily, it undergoes numerous challenges, such as physical injury, chemical insults, and UV radiation. The significance of repairing breaches of this barrier as quickly as possible is especially highlighted in chronic nonhealing wounds, including those associated with diabetes, which affects one in ten people worldwide, with vascular disease and aging. Dying cells in the wound perpetuate inflammation, contribute to dysregulated tissue repair, and increase the risk of infection. Cutaneous wound healing typically consists of four phases, which are hemostasis, inflammation, growth, re-epithelialization, and remodeling. In this chapter, we will discuss how various cell types, immune and nonimmune cells that reside in the epidermis and dermis, die from apoptosis and how efficient dead cell clearance by professional and nonprofessional phagocytes contributes to effective skin wound healing. We will also consider a range of cell death modalities, such as ferroptosis, necroptosis, pyroptosis, and others, and present our current knowledge about their contribution to skin injury and repair.
Indexed as
Identifiers
41004082What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.