Evidence mapPaperPMID 41009334Full record

SynthesisInternational journal of molecular sciences2025

Reduced Systemic Levels of Bile Acids in Individuals with Coronary Artery Disease: Insights from a Systematic Review.

Víctor Manuel López Espinosa, Francisco J Amaro-Gahete, Francisco J Osuna-Prieto

Abstract readSystematic Review
In one paragraph

Synthesis in International journal of molecular sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Víctor Manuel López EspinosaCardiology Department, University Hospital Virgen de las Nieves, 18014 Granada, Spain.ORCID 0009-0004-0743-4263
Francisco J Amaro-GaheteDepartment of Physiology, Faculty of Medicine, Sport and Health University Research Institute (iMUDS), University of Granada, 18010 Granada, Spain.ORCID 0000-0002-7207-9016
Francisco J Osuna-PrietoHospital Universitario Joan XXIII de Tarragona, Institut d'Investigació Sanitària Pere Virgili (IISPV), 43005 Tarragona, Spain.ORCID 0000-0002-6546-9534

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Bile acids (BAs) play a key role in cholesterol metabolism and inflammation. Although altered circulating BA profiles have been reported in cardiometabolic disorders such as type 2 diabetes (T2D) and obesity, their relationship with coronary artery disease (CAD) remains poorly understood. We conducted a systematic review of human studies searching PubMed, Web of Science, and Scopus, assessing circulating BA concentrations in adults with angiographically confirmed CAD compared to non-CAD (NCAD) controls. Risk of bias was evaluated using the Newcastle-Ottawa Scale. From 2782 records, four observational studies met the inclusion criteria. All reported lower circulating BA concentrations in individuals with CAD compared to NCAD controls, with differences ranging from -5.4% to -52.8%. Two studies found a significant inverse association between BA levels and CAD. One study reported lower BA levels only in CAD in men, while another found the reduction more pronounced in individuals with T2D. However, all studies were observational, and most lacked adjustment for confounders such as sex and age. Current evidence suggests that lower circulating BA levels are linked to CAD and may be influenced by sex and T2D status. Further mechanistic and prospective studies are needed to clarify the relevance and directionality of this association.

Indexed as

Bile Acids and SaltsCoronary Artery DiseaseDiabetes Mellitus, Type 2FemaleHumansMaleBile Acids and Saltsatherosclerosisbiomarkerscardiovascular risklipid metabolism

Identifiers

PMID41009334
PMCPMC12469891

What Socratic holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.