Evidence map›Paper›PMID 41010003›Full record

ReviewGenes2025

The Impact of Genetic Polymorphisms on the Clinical Efficacy of Azole Antifungals.

Hareesh Singam, Sherif Mossad

Abstract readReview
In one paragraph

Review in Genes, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Hareesh SingamDepartment of Infectious Diseases, Section of Transplant Infectious Diseases, Cleveland Clinic Foundation, Cleveland, OH 44195, USA.
Sherif MossadDepartment of Infectious Diseases, Section of Transplant Infectious Diseases, Cleveland Clinic Foundation, Cleveland, OH 44195, USA.ORCID 0000-0002-2394-1423

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Azoles are the primary agents for antifungal activity in clinical medicine due to their broad-spectrum efficacy and favorable safety profiles compared to older agents. Triazoles, including fluconazole, itraconazole, voriconazole, posaconazole, and isavuconazole, have varied pharmacokinetic and pharmacodynamic properties. This is due to various polymorphisms in hepatic enzymes, necessitating genotype-guided dosing and therapeutic drug monitoring (TDM) to optimize treatment outcomes. This review highlights the clinical relevance of pharmacogenomics in azole therapy, particularly the role of cytochrome P450 (CYP450) enzyme polymorphisms in influencing drug levels, efficacy, and toxicity. Understanding these genetic and metabolic factors is essential for personalized antifungal treatment strategies, improving patient safety and therapeutic outcomes.

Indexed as

Antifungal AgentsAzolesCytochrome P-450 Enzyme SystemMycosesPolymorphism, GeneticDrug MonitoringHumansPharmacogeneticsTriazolesAntifungal AgentsAzolesCytochrome P-450 Enzyme SystemTriazolesCYP2C19cytochrome P450itraconazoleposaconazoletherapeutic drug monitoringvoriconazole

Identifiers

PMID41010003
PMCPMC12469605

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.