Evidence map›Paper›PMID 41012800›Full record

ReviewVeterinary sciences2025

Comparative Genetics of Canine and Human Cancers.

Richard Curtis Bird, Bruce F Smith

Abstract readReview
In one paragraph

Review in Veterinary sciences, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Richard Curtis BirdDepartment of Pathobiology, College of Veterinary Medicine, Auburn University, Auburn, AL 36849, USA.
Bruce F SmithScott-Ritchey Research Center and Department of Pathobiology, College of Veterinary Medicine, Auburn University, Auburn, AL 36849, USA.ORCID 0000-0001-8523-0355

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Domestic dogs develop cancer at rates similar to, or greater than, humans and suffer from cancer types that also reflect several similar common types of human disease. Canine cancers have, as a consequence, been investigated as a model of human cancer that is particularly attractive because it is an intermediate sized animal model with an intact immune system that develops spontaneous disease. This discipline has had extraordinary consequences for canine medicine as well, promoting the exploration of the genetics of canine cancers and the development of new therapies designed to be translated to humans but that also benefit the dogs themselves. We review the gene defects found in the most well-studied canine cancers and the new therapeutic approaches such investigations have promoted.

Indexed as

cancercaninegeneshumanmodel systemspontaneous diseasetherapy

Identifiers

PMID41012800
PMCPMC12474490

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.