Evidence mapPaperPMID 41049266Full record

ReviewMedComm2025

Breast Cancer: Molecular Pathogenesis and Targeted Therapy.

Md Abdus Samad, Iftikhar Ahmad, Mohammad Rashid Khan, Mohd Suhail, Torki A Zughaibi, Fahad A Al-Abbasi, Khaled A Alhosaini, Mohd Shahnawaz Khan, Ajoy Kumer, Shams Tabrez

Abstract readReview
In one paragraph

Review in MedComm, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed, 1 pooled it
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Article
  4. Review
  5. Article
  6. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors.

Md Abdus SamadDepartment of Biochemistry Faculty of Science King Abdulaziz University Jeddah Saudi Arabia.
Iftikhar AhmadDepartment of Biochemistry Faculty of Science King Abdulaziz University Jeddah Saudi Arabia.
Mohammad Rashid KhanDepartment of Pharmacology and Toxicology College of Pharmacy King Saud University Riyadh Saudi Arabia.
Mohd SuhailKing Fahd Medical Research Center King Abdulaziz University Jeddah Saudi Arabia.
Torki A ZughaibiKing Fahd Medical Research Center King Abdulaziz University Jeddah Saudi Arabia.
Fahad A Al-AbbasiDepartment of Biochemistry Faculty of Science King Abdulaziz University Jeddah Saudi Arabia.
Khaled A AlhosainiDepartment of Pharmacology and Toxicology College of Pharmacy King Saud University Riyadh Saudi Arabia.
Mohd Shahnawaz KhanProtein Research Chair Department of Biochemistry College of Sciences King Saud University Riyadh Saudi Arabia.
Ajoy KumerDepartment of Chemistry College of Arts and Sciences International University of Business Agriculture & Technology (IUBAT) Dhaka Bangladesh.
Shams TabrezKing Fahd Medical Research Center King Abdulaziz University Jeddah Saudi Arabia.ORCID https://orcid.org/0000-0003-4550-415X

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Breast cancer (BC) is the most prevalent cancer in women and remains the leading cause of cancer-related mortality globally. Its development is influenced by multiple factors, including genetics, environmental, aging, and modulation of various signaling pathways. The heterogeneity of BC together with the emergence of treatment resistance and recurrence have prompted researchers to explore and develop new therapeutic approaches. Recently, oncology research has primarily focused on the development of targeted therapies against molecular abnormalities in BC. These therapies include monoclonal antibodies, tyrosine kinase inhibitors, antibody-drug conjugates, PI3K/Akt/mTOR pathway inhibitors, CDK 4/6 inhibitors, PARP inhibitors, antiangiogenic agents, and various other targeted drugs. Immunomodulatory strategies, including immune checkpoint inhibitors (anti-PD-1/PD-L1), CTLA-4 blockers, adoptive T-cell therapy, and cancer vaccines, stimulate immune response against cancer cells. Epigenetic therapies like DNMT and HDAC inhibitors have also shown promise in BC treatment. This review highlights how innovative approaches like targeting intratumoral heterogeneity, liquid biopsy for resistance mutation detection, bypass mechanisms (

Indexed as

artificial intelligencebreast cancer subtypesPARP inhibitorsPI3K/Akt/mTOR inhibitorstargeted therapytumor microenvironment

Identifiers

PMID41049266
PMCPMC12495454

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.