ArticleFrontiers in medicine2025
Risk factors and clinical outcomes of severe postoperative hypoxaemia following surgical repair of Stanford type A aortic dissection: a retrospective cohort study.
Article in Frontiers in medicine, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.
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Abstract
Objective: This study aimed to investigate independent risk factors of severe postoperative hypoxaemia in patients with type A aortic dissection (TAAD), for providing a reference for clinical healthcare professionals to identify high-risk patients early and formulate postoperative severe hypoxaemia prevention and intervention plans. Methods: This was a retrospective, observational study, patients with Stanford TAAD who underwent aortic surgery ( Results: Of the 755 patients with postoperative TAAD, 192 (25.43%) experienced severe postoperative hypoxaemia. Multivariable logistic regression analysis revealed body mass index (BMI) (odds ratio [OR] = 1.137), white blood cell count (WBC) (OR = 1.068), hypertension (OR = 2.693), chronic kidney disease (CKD) (OR = 2.767), highest lactate level (OR = 1.094) as independent predictors of severe postoperative hypoxaemia. Femoral artery cannulation (OR = 0.533) was a protective factor against severe postoperative hypoxaemia. Severe hypoxaemia increased hospitalization costs, prolonged mechanical ventilation duration, and extended intensive care unit and hospital stay durations. Conclusion: The high severe hypoxaemia prevalence after TAAD is associated with poor clinical outcomes. Preoperative BMI, WBC, hypertension, CKD and intraoperative highest lactate level were risk factors for severe postoperative hypoxaemia. Clinical healthcare professionals should closely monitor high-risk patients, intensify oxygenation index surveillance, and intervene promptly to minimize hypoxaemia and improve patient prognosis.
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