ReviewJournal of experimental botany2026
Linking redox and calcium signaling via thioredoxins.
Review in Journal of experimental botany, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
5 citing papers in PubMed.
- Multiscale strategies to enhance plant resilience under climate change.Journal of experimental botany · 2026Article
- GhSTH-2 Integrates Host Redox Signalling and Effector Recognition and Confers Resistance Against Verticillium dahliae in Cotton.Molecular plant pathology · 2026Article
- Understanding the Role of Durum Wheat ThioredoxinPlants (Basel, Switzerland) · 2026Article
- Molecular mechanisms of plant freezing tolerance: from cold signal perception to adaptive responses.Frontiers in plant science · 2026Review
- Emerging roles of silicon in plant signaling networks and microbiome dynamics under salt stress.Frontiers in plant science · 2026Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
Abstract
Reactive oxygen species (ROS) and calcium (Ca2+) are well-established second messengers in plants that transmit signals from an initial stimulus to initiate downstream cellular processes, and play crucial roles in plant development and responses to adverse environmental conditions. Upon perception of stimuli, transient increases in ROS and cytosolic free calcium concentration ([Ca2+]cyt) act as early signals. Later, Ca2+ signals regulate ROS homeostasis by controlling ROS generation, but reciprocally, ROS can modulate Ca2+ signaling via post-translational modifications (PTMs) in specific proteins involved in the transport, sensing, and transmission of the Ca2+ signal. Thioredoxins (TRXs) act as ROS scavengers, being able to revert the oxidized, nitrosated, persulfided, and glutathionylated forms of specific proteins, acting as a reversible PTM mechanism that contributes to fine-tune protein function. In this review, we summarize the crosstalk between Ca2+ and ROS signaling, focusing on the involvement of TRXs.
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Identifiers
What Socratic holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.