Evidence mapPaperPMID 41153608Full record

ReviewBiomedicines2025

Role of Adipokines Chemerin, Visfatin, and Omentin in Obesity and Their Inflammatory and Metabolic Implications.

Wilson José S Pedro, Flávio V Barbosa Júnior, Fernanda N B R Alves, Lenita V Braga, Larissa R Alves, João Pedro R Afonso, Iranse O Silva, Carlos Hassel M Silva, Sergio Vencio, Paolo Capodaglio and 2 more

Abstract readReview
In one paragraph

Review in Biomedicines, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 7 papers.

0numbers the graph read from it
0cells of the map it votes in
7citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

7 citing papers in PubMed.

  1. Trial
  2. Review
  3. Review
  4. Circulating Omentin Levels in Heart Failure: A Case-Control Study.International journal of molecular sciences · 2026
    Article
  5. Review
  6. Review
  7. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors.

Wilson José S PedroGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.
Flávio V Barbosa JúniorGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.
Fernanda N B R AlvesGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.
Lenita V BragaGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.
Larissa R AlvesGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.
João Pedro R AfonsoGraduate Program in Human Movement and Rehabilitation (PPGMHR), Evangelical University of Goiás (UniEVANGELICA), 75083-515 Anapolis, Brazil.
Iranse O SilvaGraduate Program in Human Movement and Rehabilitation (PPGMHR), Evangelical University of Goiás (UniEVANGELICA), 75083-515 Anapolis, Brazil.ORCID 0000-0003-2692-1548
Carlos Hassel M SilvaGraduate Program in Human Movement and Rehabilitation (PPGMHR), Evangelical University of Goiás (UniEVANGELICA), 75083-515 Anapolis, Brazil.
Sergio VencioGraduate Program in Human Movement and Rehabilitation (PPGMHR), Evangelical University of Goiás (UniEVANGELICA), 75083-515 Anapolis, Brazil.
Paolo CapodaglioOrthopaedic Rehabilitation Unit and Research Lab for Biomechanics, Rehabilitation and Ergonomics, Ospedale San Giuseppe, Istituto Auxologico Italiano, IRCCS, 28824 Piancavallo di Oggebbio, Italy.ORCID 0000-0003-3719-8789
Luis V F OliveiraGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.ORCID 0000-0002-3852-9415
Wilson R Freitas JúniorGraduate Program in Health Sciences at the Faculty of Medical Sciences of Santa Casa de São Paulo (PPGCS), 01224-001 Sao Paulo, Brazil.ORCID 0000-0002-8394-3842

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Obesity is a multifactorial disease with endocrine, metabolic, and inflammatory underpinnings, leading to numerous comorbidities and increased mortality. This has driven research into adipose tissue's role as an endocrine organ that secretes adipokines. This review critically analyzes three of these adipokines: chemerin, omentin-1, and visfatin. Chemerin and omentin-1 have well-defined roles as pro- and anti-inflammatory mediators, respectively. However, the function of visfatin remains controversial, with conflicting data regarding its role in glucose metabolism and inflammation. This conflicting evidence highlights an urgent need for standardized assays and population-specific studies to clarify its true function. We conclude that while chemerin and omentin-1 represent promising targets, the ambiguity surrounding visfatin limits its current clinical utility, and resolving these knowledge gaps is essential for developing effective biomarkers and therapies for obesity and its comorbidities.

Indexed as

adipokinesbiomarkerschemerininflammationobesityomentinvisfatin

Identifiers

PMID41153608
PMCPMC12562133

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.