Evidence mapPaperPMID 41196683Full record

ArticleCell reports2025

Molecular and functional asymmetry in Cckar-expressing vagal sensory neurons.

Hailey F Welch, Ishwarya Sankaranarayanan, Veronica M Hong, Haniya Qavi, Khadijah Mazhar, Benedict J Kolber, Theodore J Price, Catherine A Thorn

Abstract read
In one paragraph

Article in Cell reports, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed.

  1. Article
  2. Article
  3. Vagal GABAergic signaling in autonomic control of cardiometabolic function.American journal of physiology. Cell physiology · 2026
    Review
  4. Glucagon-Like Peptide-1 Targets in the Human Nodose Ganglion.The Journal of comparative neurology · 2026
    Article
  5. Review
  6. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

8 authors.

Hailey F WelchDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Ishwarya SankaranarayananDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA; Center for Advanced Pain Studies, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Veronica M HongDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA; Center for Advanced Pain Studies, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Haniya QaviDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Khadijah MazharDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA; Center for Advanced Pain Studies, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Benedict J KolberDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA; Center for Advanced Pain Studies, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Theodore J PriceDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA; Center for Advanced Pain Studies, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA.
Catherine A ThornDepartment of Neuroscience, The University of Texas at Dallas, 800 W. Campbell Rd., Richardson, TX 75080, USA. Electronic address: catherine.thorn@utdallas.edu.

Funding

Translation Control of Pain PlasticityR01NS065926 · NINDS · UNIVERSITY OF TEXAS DALLAS · PI Theodore J. Price · 2023 to 2023
$537k
Impact of Amygdala Lateralization on Processing and Modulation of Bladder PainR01DK115478 · UNIVERSITY OF TEXAS DALLAS · 2025 to 2025
$471k
Sigma 2 Receptor (TMEM97): Investigating the Peripheral Role of this Novel Therapeutic Target for PainF31NS129269 · NINDS · UNIVERSITY OF TEXAS DALLAS · 2024 to 2025
$86k
NIDA NIH HHS R21 DA055166NIDDK NIH HHS R01 DK115478NINDS NIH HHS F31 NS129269NINDS NIH HHS R01 NS065926
6 · The paper itself

Abstract

The vagus nerves are important carriers of sensory information from the viscera to the brain. Emerging evidence suggests that sensory signaling through the right, but not the left, vagus nerve evokes striatal dopamine release and reinforces appetitive behaviors. However, the extent to which differential gene expression within vagal sensory neurons may underlie this asymmetric reward-related signaling is unknown. Here, we use single-nucleus RNA sequencing, in situ hybridization, and calcium imaging to identify a single cluster of neurons, defined by co-expression of Chrna3 (nicotinic acetylcholine receptor subunit 3) and Cckar (cholecystokinin 1 receptor), that is preferentially expressed in the right nodose ganglia of rats and mice. This neuronal population also expresses several genes implicated in digestive signaling, including Glp1r and Sctr, consistent with a gut-innervating subtype. Our results suggest that right-biased expression of this unique chemosensing vagal cell type may contribute to asymmetric encoding of interoceptive rewards by the vagus nerves.

Indexed as

Receptor, Cholecystokinin ASensory Receptor CellsVagus NerveAnimalsMaleMiceMice, Inbred C57BLNodose GanglionRatsRats, Sprague-DawleyReceptor, Cholecystokinin AasymmetrycholecystokininCP: Neurosciencegut-brain rewardinteroceptionlateralizationnodose gangliatranscriptomicsvagus nerve

Identifiers

PMID41196683
PMCPMC12768524

What Socratic holds

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LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.