Evidence map›Paper›PMID 41201520›Full record

ArticleMolecular neurobiology2025

Puerarin Alleviates High-fat High-sugar Diet-induced Alzheimer's Disease-like Pathology and Insulin Resistance in Mice by Inhibiting p35/CDK5-mediated Mitochondrial Fission.

Hailing Lv, Ni Mao, Aiju Liu, Xiumin Zhao, Baike Zhu

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Article in Molecular neurobiology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

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4citing papers in PubMed
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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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3 · Its place in the literature

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4 citing papers in PubMed.

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Hailing LvDepartment of Neurology, Shandong Provincial Third Hospital, Shandong University, No.11 Wuyingshan Road, Tianqiao District, Jinan, 250000, Shandong, China.
Ni MaoDepartment of Neurology, The 960th Hospital of Chinese People's Liberation Army, Jinan, 250000, Shandong, China.
Aiju LiuDepartment of Neurology, Shandong Provincial Third Hospital, Shandong University, No.11 Wuyingshan Road, Tianqiao District, Jinan, 250000, Shandong, China.
Xiumin ZhaoDepartment of Neurology, Shandong Provincial Third Hospital, Shandong University, No.11 Wuyingshan Road, Tianqiao District, Jinan, 250000, Shandong, China. wusuowei6960@sina.com.
Baike ZhuDepartment of Neurology, Shandong Provincial Third Hospital, Shandong University, No.11 Wuyingshan Road, Tianqiao District, Jinan, 250000, Shandong, China. zbk6201@163.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Puerarin has demonstrated protective effects against Alzheimer's disease (AD) and diabetes. This study aimed to explore the therapeutic potential and mechanistic basis of puerarin against high-fat high-sugar (HFHS) diet-induced AD-like pathology and insulin resistance, with a specific focus on p35/cyclin-dependent kinase 5 (CDK5) signaling. SH-SY5Y cells were cultured under high glucose (HG) conditions to induce AD-like pathology, and mice were fed with an HFHS diet to establish an AD model. Both models were treated with various concentrations of puerarin. The expression and activity of p35/CDK5 were examined by Western blot and kinase assays. Rescue experiments were conducted by transfecting SH-SY5Y cells with p35 overexpression plasmids. The protein levels of amyloid-beta (Aβ)42, amyloid precursor protein (APP), and phosphorylated-Tau were assessed by immunofluorescence. The protein levels in the insulin signal-related signaling pathway were examined. Mitochondrial dysfunction mediated by CDK5/dynamin-related protein 1 (DRP1) was determined. Additionally, cognitive function in mice was evaluated using behavioral tests, including the open field test, novel object recognition test, and Morris water maze. Insulin resistance in mice was assessed using biochemical assays. Puerarin inhibited HG-induced p35/CDK5 activation in SH-SY5Y cells. It also decreased the HG-induced upregulation of Aβ42, APP, and p-Tau in SH-SY5Y cells. Moreover, puerarin ameliorated HG-induced insulin resistance and mitochondrial dysfunction in SH-SY5Y cells, as evidenced by improved insulin signaling and restored mitochondrial ultrastructure. However, p35 overexpression abrogated these protective effects. In vivo, puerarin alleviated HFHS diet-induced cognitive impairment, Aβ deposition, and Tau phosphorylation in mice. Furthermore, puerarin ameliorated HFHS diet-induced insulin resistance and mitochondrial dysfunction in mice. Puerarin ameliorated HFHS diet-induced cognitive impairment, insulin resistance, and mitochondrial dysfunction by inhibiting p35/CDK5 activity. Our findings highlight the therapeutic potential of puerarin in the management of diet-induced AD.

Indexed as

Alzheimer DiseaseCyclin-Dependent Kinase 5Diet, High-FatInsulin ResistanceIsoflavonesMitochondrial DynamicsAmyloid beta-PeptidesAnimalsCell Line, TumorGlucoseHumansMaleMiceMice, Inbred C57BLMitochondriaSignal TransductionAmyloid beta-PeptidesCdk5 protein, mouseCyclin-Dependent Kinase 5GlucoseIsoflavonespuerarinAlzheimer's diseaseHigh-fat high-sugar dietInsulin resistanceMitochondrial dysfunctionP35/CDK5Puerarin

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.