Evidence map›Paper›PMID 41209007›Full record

ReviewFrontiers in immunology2025

Immune markers for pulmonary aspergillosis in patients with chronic obstructive pulmonary disease: a narrative review.

Yuke Hu, Xingbo Wang, Hao Wu, Haibo Deng, Fei Gao, Yonghong Wu

Abstract readReview
In one paragraph

Review in Frontiers in immunology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Research trends in COPD-associated invasive pulmonary aspergillosis over the past two decades: a bibliometric and topic modelling analysis.European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology · 2026
    Review
  2. Article
  3. Association betweenFrontiers in immunology · 2026
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Yuke Hu *School of Medical and Life Sciences, Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Xingbo Wang *School of Medical and Life Sciences, Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Hao WuSchool of Medical and Life Sciences, Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Haibo DengDepartment of Respiratory Medicine, Jianyang People's Hospital, Jianyang, China.
Fei GaoSchool of Modern Chinese Medicine Industry/School of Pharmacy, Chengdu University of Traditional Chinese Medicine, Chengdu, China.
Yonghong WuDepartment of Respiratory Medicine, Jianyang People's Hospital, Jianyang, China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

COPD complicated by pulmonary aspergillosis (COPD-PA) encompasses invasive, chronic, and allergic phenotypes and is increasingly recognized as a high-burden comorbidity. Clinical recognition is often hindered by nonspecific manifestations, corticosteroid-suppressed inflammatory signs, and the suboptimal performance of serum galactomannan in non-neutropenic hosts. To define the translational utility of immune biomarkers in this population, evidence was synthesized from international guidelines and contemporary studies in COPD-enriched cohorts, and performance was appraised across diagnostic, monitoring, and prognostic domains. Convergent findings indicate that bronchoalveolar-lavage galactomannan facilitates early diagnosis; serum galactomannan indices stratify risk during exacerbations; Aspergillus-specific IgG supports rule-in for invasive and chronic disease; and pentraxin-3 adds prognostic information. Cytokines central to COPD-PA pathobiology, including interleukin-1β, interleukin-6, interleukin-8, and interleukin-17, provide adjunctive diagnostic value, whereas (1→3)-β-D-glucan shows limited specificity. On this basis, a three-tier framework was developed that classifies biomarkers as clinically validated, mechanistically promising, or exploratory, and this framework was translated into subtype-tailored panels and decision rules that favor either-positive criteria for screening and both-positive criteria for confirmation. It is concluded that immune biomarkers complement microbiology and imaging, expand access when bronchoscopy or biopsy is not feasible, and enable longitudinal risk stratification. Priorities include COPD-specific thresholds, assay standardization, and multicenter validation, with particular emphasis on chronic pulmonary aspergillosis and allergic bronchopulmonary aspergillosis. Biomarker-guided immunomodulation may benefit selected phenotypes but requires rigorous evaluation before clinical adoption.

Indexed as

AspergillusPulmonary AspergillosisPulmonary Disease, Chronic ObstructiveBiomarkersCytokinesGalactoseHumansMannansPrognosisSerum Amyloid P-ComponentBiomarkersCytokinesgalactomannanGalactoseMannansSerum Amyloid P-Componentchronic obstructive pulmonary diseasediagnostic algorithmgalactomannanimmune biomarkerspulmonary aspergillosis

Identifiers

PMID41209007
PMCPMC12589061

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.