Evidence map›Paper›PMID 41211649›Full record

ArticleJournal of the American Heart Association2026

Electroacupuncture Alleviates Brain Injury Through Vagus Nerve Activation and Gut Microbiota in a Rat Model of Ischemic Stroke.

Qian-Kun Zhao, Yue-Xin Ning, Tian-Ce Xu, Zi-Ai Zhao, Yi-Feng Pei, Jing-Yuan Niu, Xian-Dong Li, Hui-Sheng Chen

Abstract read
In one paragraph

Article in Journal of the American Heart Association, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed, 2 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. Pooled it
  3. Article
  4. Review
  5. Review
  6. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors.

Qian-Kun ZhaoDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0009-0004-6130-6115
Yue-Xin NingDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0009-0002-1805-8705
Tian-Ce XuDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0009-0004-5855-8426
Zi-Ai ZhaoDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.
Yi-Feng PeiDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0000-0003-2372-3343
Jing-Yuan NiuDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.
Xian-Dong LiDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0009-0003-5351-6457
Hui-Sheng ChenDepartment of Neurology General Hospital of Northern Theater Command Shenyang 110016 China.ORCID 0000-0002-7486-1992

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundEmerging evidence implicates gut microbiota dysbiosis in exacerbating stroke pathogenesis via the gut-brain axis, suggesting novel therapeutic targets. While electroacupuncture (EA) demonstrates anti-inflammatory effects through vagus nerve activation, its neuroprotective mechanisms via vagus nerve-microbiota crosstalk remain unexplored.

methodsRats with middle cerebral artery occlusion received daily ST36 (Acupoint Zusanli) EA for 1 to 7 days postischemia. Subdiaphragmatic vagotomy and fecal microbiota transplant were implemented to validate pathway specificity. Multimodal assessments included longitudinal neurological scoring, infarct volume, systemic/neuroinflammatory profiling (enzyme-linked immunosorbent assay, immunohistochemistry), intestinal fucosylation dynamics (quantitative polymerase chain reaction, lectin staining), and 16S ribosomal RNA sequencing of gut microbiota.

resultsEA significantly improved neurological outcomes and reduced infarct volumes at 3 to 7 days after middle cerebral artery occlusion (versus controls), which was abolished by vagotomy. Mechanistically, EA restored gut barrier integrity through vagus-dependent upregulation of fucosyltransferase 2 (Fut2)-driven epithelial α1,2-fucosylation, enhancing mucin 2+ goblet cell density and tight junction protein expression (ZO-1/occludin/claudin-1). Concurrent microbiota shifts included Lactobacillales/Bacteroidales enrichment (linear discriminant analysis >4.0) and pathobiont suppression, which was reversed by vagotomy. Crucially, fecal microbiota transplant from EA-treated donors replicated neuroprotection in germ-free recipients, achieving 33% infarct reduction and 30% survival improvement (

conclusionsEA at ST36 produced neuroprotection through activating vagal efferent pathways to orchestrate intestinal mucosal repair via Fut2-mediated fucosylation, which reshape microbial ecosystems and attenuate neuroinflammation. These findings establish a previously unrecognized vagus nerve-gut-brain axis mechanism for stroke recovery, positioning microbiota-directed neuromodulation by EA as a translatable therapeutic strategy.

Indexed as

ElectroacupunctureGastrointestinal MicrobiomeInfarction, Middle Cerebral ArteryIschemic StrokeVagus NerveAnimalsBrainDisease Models, AnimalFecal Microbiota TransplantationMaleRatsRats, Sprague-Dawleyelectroacupuncturegut‐brain axisgut microbiotaischemic strokevagus nerve

Identifiers

PMID41211649
PMCPMC12909005

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.