Vitamin D and Fracture Healing.

Md S Zaman, M I Ullah

Abstract readReview

In one paragraph

Review in Advances in experimental medicine and biology, 2026. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it

0cells of the map it votes in

0citing papers in PubMed

–field-weighted citation impact

1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Md S ZamanDepartment of Biological Sciences, Alcorn State University, Lorman, MS, USA. zaman@alcorn.edu.

M I UllahDivision of Internal Medicine and Hypertension, University of Mississippi Medical Center, Jackson, MS, USA.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Maintaining bone health involves complex interactions among nutrients, hormones, and physiological mechanisms, with vitamin D playing a pivotal role. Vitamin D is essential for the absorption of calcium and phosphate, which are critical minerals for bone development, maintenance, and repair. Disruptions in mineral ion homeostasis can lead to conditions such as hypocalcemia, osteoporosis, osteomalacia, and rickets, which are prevalent worldwide. Inadequate vitamin D impairs bone health, increasing the risk of fractures and catabolic bone diseases. This chapter elaborates on the synthesis, activation, and function of vitamin D, highlighting its essential role in bone health, from supporting growth in children to maintaining bone strength in adults. It also elaborates on how vitamin D enhances healing after a fracture by regulating the immune response, inflammation, and mineralization process.

Indexed as

Bone and BonesFracture HealingFractures, BoneVitamin DVitamin D DeficiencyAnimalsHumansVitamin DBone growthCalcium regulationCholecalciferolErgocalciferolFracture healingPhosphate regulationVitamin D activation

Identifiers

PMID41219601

DOI10.1210/JC.2011-0385

What Socratic holds

Textmetadata

Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.