Evidence mapPaperPMID 41224303Full record

ArticleBMJ open2025

Metabolic dysfunction-associated steatotic liver disease and colorectal neoplasms risk: a global propensity score-matched retrospective cohort study.

Mohammad Aldiabat, Ali Osman, Malek Ayoub, Mahmoud Y Madi, Kamran Qureshi, Wing-Kin Syn

Abstract readMulticenter Study
In one paragraph

Article in BMJ open, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Mohammad AldiabatDepartment of Medicine, Washington University in St Louis, St. Louis, Missouri, USA Mohameddiabat88@gmail.com.ORCID http://orcid.org/0000-0002-4536-2799
Ali OsmanDepartment of Medicine, Washington University in St Louis, St. Louis, Missouri, USA.
Malek AyoubDepartment of Medicine, Washington University in St Louis, St. Louis, Missouri, USA.
Mahmoud Y MadiDivision of Gastroenterology & Hepatology, Saint Louis University, St. Louis, Missouri, USA.
Kamran QureshiDivision of Gastroenterology & Hepatology, Saint Louis University, St. Louis, Missouri, USA.
Wing-Kin SynDivision of Gastroenterology & Hepatology, Saint Louis University, St. Louis, Missouri, USA.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

objectivesTo evaluate the association between metabolic dysfunction-associated steatotic liver disease (MASLD), metabolic-associated steatohepatitis (MASH), and the risk of colorectal cancer (CRC) and benign colorectal neoplasms (BCN), and to explore whether liver fibrosis/cirrhosis modifies these associations.

designRetrospective cohort study with 1:1 propensity score matching.

settingGlobal, multicentre real-world analysis using deidentified electronic health records from over 130 healthcare organisations in the TriNetX Global Collaborative Network.

participantsHospitalised adults aged 45-75 years between October 2019 and October 2024. Patients with prior diagnoses of colorectal neoplasia or other chronic liver diseases were excluded. Final matched cohorts included 138 902 MASLD and non-MASLD patients, 3715 MASH and non-MASH patients, and 1312 MASH patients with and without fibrosis. PRIMARY AND SECONDARY OUTCOME MEASURES: Primary outcomes: Incidence of CRC and BCN. SECONDARY OUTCOME: Combined incidence of CRC and BCN. Outcomes were assessed with and without controlling for metabolic risk factors using Cox proportional hazards models.

resultsMASLD was associated with increased risks of CRC (HR 2.71, 95% CI 2.29 to 3.20) and BCN (HR 2.50, 95% CI 2.38 to 2.63), both p<0.001. MASH patients had a 5-fold higher risk of CRC (HR 5.03, 95% CI 1.43 to 17.72, p=0.005) and nearly 2-fold risk of BCN (HR 1.91, 95% CI 1.38 to 2.67, p<0.001). No significant differences in CRC or BCN risk were observed between MASH patients with versus without fibrosis/cirrhosis.

conclusionsMASLD and MASH are independent risk factors for CRC and BCN, irrespective of metabolic comorbidities. Fibrosis/cirrhosis does not significantly influence CRC risk. These findings support the need to revisit CRC screening guidelines for patients with MASLD/MASH. Further prospective studies are warranted to explore underlying mechanisms and evaluate preventative interventions.

Indexed as

Colorectal NeoplasmsFatty LiverAgedFemaleHumansIncidenceLiver CirrhosisMaleMiddle AgedPropensity ScoreProportional Hazards ModelsRetrospective StudiesRisk FactorsEarly Detection of CancerEPIDEMIOLOGYGASTROENTEROLOGYGastrointestinal tumoursHepatobiliary disease

Identifiers

PMID41224303
PMCPMC12612761

What Socratic holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.