Evidence map›Paper›PMID 41233763›Full record

ArticleBMC genomics2025

RNA sequencing analysis in chicken spleen infected with Newcastle disease virus reveals genotype-specific immune response.

Haile Berihulay, Wei Luo, Chuxiao Lin, Ainong Lao, Endashaw Jebessa, Xian Zou, Jian Ji, Hao Qu, Manshan Cai, Dingming Shu and 1 more

Abstract read
In one paragraph

Article in BMC genomics, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors.

Haile Berihulay *State Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Wei Luo *State Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Chuxiao LinState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Ainong LaoState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Endashaw JebessaState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Xian ZouState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Jian JiState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Hao QuState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Manshan CaiState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Dingming ShuState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China.
Chenglong LuoState Key Laboratory of Swine and Poultry Breeding Industry, Guangdong Provincial Key Laboratory of Animal Breeding and Nutrition, Institute of Animal Science, Guangdong Academy of Agricultural Sciences, Guangzhou, China. chenglongluo1981@163.com.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundGlobally, the Newcastle disease virus (NDV) is a seriously important pathogen and results in substantial economic losses in the poultry industry. To mitigate this pathogen, the poultry industry has employed intensive breeding programs aimed at selecting genetic Lines that promote resistance and enhance immune response. Resistance to infection occurs at multiple levels and involves genotype-specific polymorphisms in the host. To date, the influence of genetic variations on the immune response within the same chicken Lines at the genotype-specific level is not fully understood. Therefore, it is important to understand the host genetic resistance that plays a role after NDV infection. This study aimed to investigate genotype-specific immune responses in chicken spleens following NDV infection at 7 days post-infection (7dpi), Samples were analyzed for differentially expressed genes (DEGs) using high-throughput RNA sequencing approaches.

resultsTwelve cDNA Libraries from LNH chicken spleens were compared with those of NDV-infected birds across the AA and GG genotypes. A total of 9886 genes were compared, of which 1348 were upregulated (833) and downregulated (515). Of these, 552 genes, with 415 upregulated and 137 downregulated genes, were differentially expressed (DE) between the two pairwise comparison groups: Control_AA vs. Immune_AA (AA group) and Control_GG vs. Immune_GG (GG group). Higher number of differentially expressed genes (407 DEGs), with 349 upregulated and 58 downregulated genes, were identified in chickens with the GG genotype, whereas 142 genes, with 63 upregulated and 79 downregulated genes, were observed in chickens with the AA genotype. In this study, a set of seven candidate genes (LECT1, MR1, MYH3, USH1C, NMU, BG8, and CCN3) was identified associated with the immune response in chickens. Further analysis revealed that DEGs in the spleen were enriched in various signaling pathways, such as neuroactive ligand-receptor interactions, tight junctions, and glycolysis/gluconeogenesis pathways, suggesting their potential role in immune resilience.

conclusionsRNA-seq analysis revealed immune-related genes and pathways involved in the immune response in the chicken spleen. The results indicate that chickens with the GG genotype exhibited higher levels of differentially expressed genes (DEGs), potentially linked to NDV responses. Our findings also offer valuable transcriptomic insights for the comparative analysis of molecular mechanisms underlying immune response in LNH chickens and have practical implications for enhancing chicken breeding programs.

Indexed as

ChickensNewcastle DiseaseNewcastle disease virusPoultry DiseasesSpleenAnimalsGene Expression ProfilingGenotypeSequence Analysis, RNATranscriptomeChickenGenotypeImmune responseNewcastle diseaseRNA-seqSpleen

Identifiers

PMID41233763
PMCPMC12613709

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.