Evidence mapPaperPMID 41265404Full record

ArticleEnvironmental analysis, health and toxicology2025

Evaluation of the divergent effects of pyrethroid- and D-allethrin-based coil fumes in male Wistar rats.

Jelili Abiodun Badmus, John Olabode Fatoki, Adebayo Lawrence Adedeji, Ikeola Khalilat Mustapha, Blessing Chioma Ovili, Oyekunle Olufemi Oyedijo, Tolulope Fiyinfolu Olabode, Kemisola Deborah Joseph, Emmanuel Olusola Yawson, Donavon Charles Hiss

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Article in Environmental analysis, health and toxicology, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

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4 · The record

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5 · Who and what money

Authors and funding

10 authors.

Jelili Abiodun BadmusDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
John Olabode FatokiDepartment of Environmental Health Science, College of Health Sciences, Osun State University, Osogbo, Osun State, Nigeria.
Adebayo Lawrence AdedejiDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Ikeola Khalilat MustaphaDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Blessing Chioma OviliDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Oyekunle Olufemi OyedijoDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Tolulope Fiyinfolu OlabodeDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Kemisola Deborah JosephDepartment of Biochemistry, Ladoke Akintola University of Technology, Ogbomoso, Oyo State, Nigeria.
Emmanuel Olusola YawsonDepartment of Anatomy, Redeemers University, Ede Osun State, Nigeria.
Donavon Charles HissDepartment of Biomedical Sciences, University of the Western Cape, Bellville, Cape Town, South Africa.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mosquito coil fume is a cheap and commonly used method of reducing malaria incidence in third-world countries. The effects of fumes from pyrethroid and D-allethrin-based mosquito coils available in the Nigerian market were assessed in male Wistar rats. The rats were exposed to the insecticide fumes for 7, 14, and 21 days, while another group served as a control. The experiment consisted of seven randomly divided groups of six weight-matched animals per group. Plasma alanine aminotransferase (ALT), aspartate aminotransferase (AST), lactate dehydrogenase (LDH), cholesterol, phospholipids, high-density lipoprotein cholesterol (HDL-C), and high-density lipoprotein phospholipid (HDL-P) were evaluated. Lung-, liver- and kidney-reduced glutathione (GSH), glutathione peroxide (GPx), glutathione-S-transferase (GST), superoxide dismutase (SOD), malondialdehyde/lipid peroxidation (MDA) and lipid hydroperoxide (LOOH) were also evaluated. Histoimmunochemistry was used to assess lung p53 and Bcl-2 expressions. Pyrethroid and D-allethrin-based fumes induced a significant (p < 0.05) increase in plasma AST, LDH, cholesterol, phospholipids, and HDL-P, with a reduction of HDL-C levels. The fumes significantly and differently dysregulated antioxidant enzymes. The inhalations of the fumes induced significant (p < 0.05) increases in kidney MDA and LOOH levels, liver MDA by pyrethroid fume, and lung MDA by D-allethrin only, but lung LOOH by inhalations of both fumes. The increased expression of lung p53 and repression of Bcl-2 by both fumes were duration-dependent. The fume-induced disproportionate tissue function biomarkers, redox status, and apoptosis-related proteins. These effects are a possible panoply of divergent modes by which exposure to coil fumes can be deleterious to human health.

Indexed as

apoptosisD-allethrininsecticidemalondialdehydepyrethroidredox

Identifiers

PMID41265404
PMCPMC12890321

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.