Evidence map›Paper›PMID 41275224›Full record

ArticleBMC oral health2025

Effects of laser regulation on anti-inflammatory and osteogenic differentiation of periodontal ligament stem cells though NF-κB signaling pathway.

Rui Zhang, Xiangtao Ma, Jianing Song, Xiaoman Guo, Zeyu Chen, Taohong Liu, Mingxuan Wu

Abstract read
In one paragraph

Article in BMC oral health, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors.

Rui ZhangHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Xiangtao MaHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Jianing SongHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Xiaoman GuoHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Zeyu ChenHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Taohong LiuHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China.
Mingxuan WuHebei Key Laboratory of Stomatology, Department of Periodontology (II),, Hebei Clinical Research Center for Oral Diseases, School and Hospital of Stomatology, Hebei Medical University, Zhongshan East Road 383, Shijiazhuang, Hebei, 050017, People's Republic of China. wumingxuan@hebmu.edu.cn.

Funding

the Department of Education of Hebei Province ZD20220007the Department of Finance of Hebei Province ZF2023013
6 · The paper itself

Abstract

backgroundIn periodontitis, the classical inflammatory NF-κB signaling pathway is activated, which promotes alveolar bone resorption and inhibits osteogenic differentiation of periodontal ligament stem cells. Low-energy laser therapy can reduce inflammation and promote healing. This in vitro study aimed to investigate the effects of different low-energy lasers on inflammation and osteogenic differentiation of human periodontal ligament stem cells (hPDLSCs) under inflammatory conditions, and to determine whether these effects are associated with the NF-κB signaling pathway at both the protein and gene levels.

methodsHealthy premolars or third molars were collected during oral and maxillofacial surgery, and periodontal ligament tissues were obtained. hPDLSCs were cultured using the tissue block method and identified by osteogenic differentiation potential and flow cytometry. Based on treatment conditions, the cells were divided into five groups (using abbreviations from key elements of the grouping): control group (C, control), lipopolysaccharide group (L, LPS), LPS + Nd:YAG laser group (N, Nd:YAG), LPS + Er:YAG laser group (E, Er:YAG), and LPS + semiconductor laser group (D, semiconductor). Enzyme-linked immunosorbent assay (ELISA), quantitative real-time polymerase chain reaction (qRT-PCR), alizarin red staining, and immunofluorescence (IF) were performed to detect inflammatory factors, osteogenic ability, and NF-κB signaling pathway activity. Fluorescence intensity of P65 was quantified using ImageJ software. Statistical analysis was conducted using SPSS 21.0.

resultsThe cultured cells were confirmed as periodontal stem cells. Statistical significance was set at P < 0.05. The CCK-8 assay showed that on day 7, the optical density (OD) values of all three laser groups were higher than those of group L (P < 0.05). Compared with group C, IL-6 and TNF-α levels in the supernatant were increased in group L (P < 0.05). After laser treatment, IL-6 and TNF-α levels in groups N, E, and D were lower than in group L (P < 0.05), with the lowest levels observed in group N. Expression of Runx-2 and OSX mRNA was higher in groups N, E, and D than in group L, with group N showing the highest expression of osteogenic genes. The fluorescence intensity of P65 in group L was significantly higher than in group C (P < 0.05), indicating increased nuclear translocation of P65. Among the laser-treated groups, group N showed the lowest nuclear P65 fluorescence intensity (P < 0.05).

conclusionsLow-energy lasers promote anti-inflammatory activity and osteogenic differentiation of hPDLSCs in LPS-induced inflammatory environments by regulating the NF-κB signaling pathway, with Nd:YAG lasers showing the strongest effect.

Indexed as

Low-Level Light TherapyNF-kappa BOsteogenesisPeriodontal LigamentStem CellsCell DifferentiationCells, CulturedFlow CytometryHumansLasers, Solid-StateLipopolysaccharidesSignal TransductionTumor Necrosis Factor-alphaLipopolysaccharidesNF-kappa BTumor Necrosis Factor-alphaLipopolysaccharideLow-energy laser therapyNF-κB signaling pathwayOsteogenic differentiationPeriodontal stem cells

Identifiers

PMID41275224
PMCPMC12640569

What Socratic holds

Textmetadata
LicenceCC BY-NC-ND
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.