Evidence map›Paper›PMID 41278461›Full record

ArticleResearch and practice in thrombosis and haemostasis2025

Stroke severity and other predictors of venous thromboembolism in stroke patients-a population-based cohort study.

Ditte Vestergaard Hansen, Nick van Es, Henrik Toft Sørensen, Jonathan M Coutinho, Nils Skajaa

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Article in Research and practice in thrombosis and haemostasis, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

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1 · What the graph read from it

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4 · The record

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5 · Who and what money

Authors and funding

5 authors.

Ditte Vestergaard HansenDepartment of Clinical Epidemiology, Aarhus University and Aarhus University Hospital, Aarhus, Denmark.
Nick van EsDepartment of Vascular Medicine, Amsterdam University Medical Center, Location University of Amsterdam, Amsterdam, the Netherlands.
Henrik Toft SørensenDepartment of Clinical Epidemiology, Aarhus University and Aarhus University Hospital, Aarhus, Denmark.
Jonathan M CoutinhoDepartment of Neurology, Amsterdam University Medical Center, Location University of Amsterdam, Amsterdam, the Netherlands.
Nils SkajaaDepartment of Clinical Epidemiology, Aarhus University and Aarhus University Hospital, Aarhus, Denmark.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Background: Venous thromboembolism (VTE) frequently occurs after stroke, particularly within the first 3 months after diagnosis. Poststroke VTE is associated with increased mortality. Stroke severity is a known predictor of adverse prognosis; however, its ability to predict VTE is unclear. Knowledge regarding other predictors of poststroke VTE is lacking. Objectives: To identify clinical predictors of poststroke VTE in the subacute and acute phases, and to assess the association between stroke severity and poststroke VTE. Methods: In a population-based cohort study, we identified patients (aged ≥18 years and without recent VTE) with first-time ischemic stroke ( Results: In the subacute phase, 1016 (0.8%) patients developed VTE after ischemic stroke, and 200 (1.2%) patients developed VTE after intracerebral hemorrhage. In univariable analyses, VTE risk was 2- to 5-fold higher in those with severe stroke, active cancer, and previous VTE. These findings were consistent for both stroke subtypes and follow-up periods. Multivariable analyses showed no substantial alterations in the estimates for stroke severity. Conclusion: Stroke severity, active cancer, and previous VTE strongly predict poststroke VTE in patients in the subacute and acute phases. These findings offer valuable insights and can be used to identify patients at elevated risk of VTE in whom extended thromboprophylaxis may be considered.

Indexed as

clinical predictorscohort studyintracerebral hemorrhageischemic strokepopulation-basedvenous thromboembolism

Identifiers

PMID41278461
PMCPMC12634850

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.