Evidence map›Paper›PMID 41298339›Full record

ReviewImmunity, inflammation and disease2025

The Role of Reactive Oxygen Species (ROS) in Periodontitis: A Potential Therapeutic Target.

Shuyu Yang, Xi Yang

Abstract readReview
In one paragraph

Review in Immunity, inflammation and disease, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. Review
  5. Article
  6. Review
  7. Review
  8. Article
  9. Article
  10. Low salivary thioredoxin-1 levels in periodontitis.World journal of clinical cases · 2026
    Article
  11. Article
  12. Review
  13. Article
  14. Review
  15. Periodontitis and MASLD: a narrative review of the direct oral-hepatic pathway.Frontiers in cellular and infection microbiology · 2026
    Review
  16. Review
  17. Review
  18. Review
  19. Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Shuyu YangStomatological Hospital, School of Stomatology, Southern Medical University, Guangzhou, China.ORCID https://orcid.org/0009-0002-4299-8844
Xi YangStomatological Hospital, School of Stomatology, Southern Medical University, Guangzhou, China.ORCID https://orcid.org/0000-0001-5222-4805

Funding

This study was supported by the Scientific research and cultivation program from the Stomatological Hospital of Southern Medical University (grant number: PY2022014).
6 · The paper itself

Abstract

backgroundROS are pivotal in maintaining periodontal tissue health due to their dual role in physiological and pathological states. At physiological levels, ROS are essential for host defense, effectively eliminating pathogenic bacteria during inflammation and sustaining the homeostasis of the periodontal microenvironment. However, excessive ROS production disrupts the balance between oxidative activity and antioxidant defenses, leading to oxidative stress (OS). This imbalance exacerbates inflammation, damages cellular components, and drives periodontal tissue destruction. Recognizing the dual role of ROS underscores the importance of studying their regulatory mechanisms, which is crucial for advancing understanding and therapeutic strategies in periodontitis management.

objectiveThis review summarizes the latest research advancements on the molecular mechanisms linking ROS to the progression of periodontitis and explores related therapeutic strategies.

resultsThis review focuses on the pivotal role and specific mechanisms of periodontal tissue destruction by excess ROS, which mediate signaling pathways such as nuclear factor kappa B (NF-κB) and mitogen-activated protein kinase (MAPK), as well as the activation of OS response-associated proteins, including thioredoxin-interacting protein (TXNIP) and mitochondrial antiviral signaling protein (MAVS). Additionally, we provide a concise summary of emerging therapeutic strategies for periodontitis, including the use of antioxidants, photodynamic therapy (PDT), and nanomaterials. Gaining a more profound comprehension of these mechanisms may pave the way for the formulation of enhanced therapeutic strategies for periodontitis.

conclusionUnderstanding the interplay between ROS and periodontal tissue destruction is essential for advancing periodontitis research. Targeting the NOD-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome with antioxidant and ROS-modulating therapies presents a promising strategy to mitigate both inflammation and OS, thereby reducing periodontal degradation.

Indexed as

PeriodontitisReactive Oxygen SpeciesAnimalsAntioxidantsHumansInflammasomesOxidative StressSignal TransductionAntioxidantsInflammasomesReactive Oxygen SpeciesantioxidantsMAPKNF‐κBNLRP3PDTperiodontitisreactive oxygen species

Identifiers

PMID41298339
PMCPMC12657125

What Socratic holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.