Evidence mapPaperPMID 41328256Full record

ReviewClinical interventions in aging2025

Aging and Intestinal Fibrosis: Mechanisms, Implications, and Therapeutic Strategies.

Yujie Shi, Han Zheng, Xiaxin Zhu, Jianyu Lv, Mi Zhou, Shuo Zhang

Abstract readReview
In one paragraph

Review in Clinical interventions in aging, 2025. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed.

  1. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors.

Yujie Shi *Department of Gastroenterology, The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, People's Republic of China.
Han Zheng *Department of Gastroenterology, The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, People's Republic of China.
Xiaxin ZhuDepartment of Gastroenterology, The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, People's Republic of China.ORCID 0000-0003-0849-3885
Jianyu LvDepartment of General Surgical Science, Gunma University, Graduate School of Medicine, Maebashi, Japan.
Mi ZhouDepartment of Gastroenterology, The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, People's Republic of China.
Shuo ZhangDepartment of Gastroenterology, The Second Affiliated Hospital of Zhejiang Chinese Medical University, Hangzhou, People's Republic of China.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

With the global aging population, the impact of aging on various organ systems is becoming increasingly significant. The gastrointestinal tract, a key site of immune activity and microbial colonization, undergoes functional decline that is closely associated with a range of intestinal and systemic diseases. While aging-related fibrosis has been extensively studied in organs such as the lungs, liver, heart, and kidneys, its role in intestinal fibrosis remains underexplored. This review discusses mechanisms by which aging may promote or increase the risk of intestinal fibrosis, including immunosenescence, cellular senescence, gut microbiota dysbiosis, and dysregulated growth factor signaling. Additionally, both traditional and emerging therapeutic strategies are summarized to guide future interventions.

Indexed as

AgingIntestinal DiseasesIntestinesCellular SenescenceDysbiosisFibrosisGastrointestinal MicrobiomeHumansImmunosenescencecellular senescencegut microbiotaimmunosenescenceTGF-β signaling pathway

Identifiers

PMID41328256
PMCPMC12665227

What Socratic holds

Textmetadata
LicenceCC BY-NC
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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the Socratic graph.